Department of Cardiology, Academic Medical Center, University of Amsterdam, Amsterdam, The Netherlands.
J Thromb Thrombolysis. 2012 Feb;33(2):143-53. doi: 10.1007/s11239-011-0667-5.
Platelet adhesion, activation and aggregation play a pivotal role in atherothrombosis. Intracoronary atherothrombosis is the most common cause of the development of acute coronary syndrome (ACS), and plays a central role in complications occurring around percutaneous coronary intervention (PCI) including recurrent ACS, procedure-related myocardial infarction or stent thrombosis. Inhibition of platelet aggregation by medical treatment impairs formation and progression of thrombotic processes and is therefore of great importance in the prevention of complications after an ACS or around PCI. An essential part in the platelet activation process is the interaction of adenosine diphosphate (ADP) with the platelet P2Y12 receptor. The P2Y12 receptor is the predominant receptor involved in the ADP-stimulated activation of the glycoprotein IIb/IIIa receptor. Activation of the glycoprotein IIb/IIIa receptor results in enhanced platelet degranulation and thromboxane production, and prolonged platelet aggregation. The objectives of this review are to discuss the pharmacological limitations of the P2Y12 inhibitor clopidogrel, and describe the novel alternative P2Y12 inhibitors prasugrel and ticagrelor and the clinical implications of the introduction of these new medicines.
血小板黏附、激活和聚集在动脉血栓形成中起着关键作用。冠状动脉内动脉粥样血栓形成是急性冠状动脉综合征(ACS)发展的最常见原因,在经皮冠状动脉介入治疗(PCI)相关并发症中起核心作用,包括复发性 ACS、与操作相关的心肌梗死或支架血栓形成。通过药物治疗抑制血小板聚集可阻碍血栓形成过程的形成和进展,因此在预防 ACS 后或 PCI 周围的并发症方面非常重要。血小板激活过程中的一个重要部分是二磷酸腺苷(ADP)与血小板 P2Y12 受体的相互作用。P2Y12 受体是涉及 ADP 刺激糖蛋白 IIb/IIIa 受体激活的主要受体。糖蛋白 IIb/IIIa 受体的激活导致增强的血小板脱颗粒和血栓素产生,以及延长的血小板聚集。本文的目的是讨论 P2Y12 抑制剂氯吡格雷的药理学局限性,并描述新型替代 P2Y12 抑制剂普拉格雷和替卡格雷及其在引入这些新药方面的临床意义。
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