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麻醉苏醒期负压性肺水肿致应激性心肌病 1 例报告

Stress-induced cardiomyopathy after negative pressure pulmonary edema during emergence from anesthesia -A case report-.

机构信息

Department of Anesthesiology and Pain Medicine, Yonsei University College of Medicine, Seoul, Korea.

出版信息

Korean J Anesthesiol. 2012 Jan;62(1):79-82. doi: 10.4097/kjae.2012.62.1.79. Epub 2012 Jan 25.

Abstract

Stress-induced cardiomyopathy (SICM) presenting as an acute myocardial dysfunction is characterized by transient left ventricular wall motion abnormality, which has been known to be associated with excessive catecholamine production caused due to various types of stress. Sympathetic hyperactivity is common during the perioperative period, and reports of SICM occurring during this period have actually increased. We present a case of SICM following negative pressure pulmonary edema due to upper airway obstruction during emergence from anesthesia. Excessive catecholamine release in response to respiratory difficulty could have been the underlying inciting factor.

摘要

应激性心肌病(SICM)表现为急性心肌功能障碍,其特征为短暂性左心室壁运动异常,这种异常已知与各种类型的应激引起的儿茶酚胺过度产生有关。围手术期交感神经活性增高很常见,实际上,在此期间发生 SICM 的报告有所增加。我们报告了一例因麻醉苏醒期上呼吸道梗阻导致负压性肺水肿后发生 SICM 的病例。对呼吸困难的过度儿茶酚胺释放可能是潜在的诱发因素。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cc4d/3272534/dc0a10e6854f/kjae-62-79-g001.jpg

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