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脊髓侧支募集导致的 B 型夹层后截瘫的恢复。

Recovery of paraplegia after type B dissection due to spinal collateral recruitment.

机构信息

Department of Cardiovascular Surgery, Centre Hospitalier Universitaire Vaudois, Lausanne, Switzerland.

出版信息

J Vasc Surg. 2012 Jul;56(1):205-7. doi: 10.1016/j.jvs.2011.12.078. Epub 2012 Feb 9.

DOI:10.1016/j.jvs.2011.12.078
PMID:22326576
Abstract

Acute paraplegia could be a symptom of aortic dissection due to sudden compromise of arterial spinal cord blood supply. Complete spontaneous neurologic recovery is possible and was observed in the present case 3 hours after symptom onset. Spontaneous spinal cord reperfusion after acute type B dissection was probably due to two main mechanisms. Reperfusion of false lumen and collateral vascular network recruitment, recently confirmed by anatomic animal studies, serve as potential explanations. Favorable evolution of acute paraplegia after aortic dissection exists, but prognosis is uncertain, probably due to individual variable anatomic distribution of spinal cord blood supply.

摘要

急性截瘫可能是由于主动脉夹层导致的动脉脊髓血供突然受损的症状。完全自发的神经恢复是可能的,在本病例中,症状出现后 3 小时即观察到。急性 B 型夹层后脊髓自发再灌注可能归因于两个主要机制。假腔再通和侧支血管网络募集,最近在解剖学动物研究中得到证实,可作为潜在的解释。急性主动脉夹层后急性截瘫的预后较好,但不确定,可能与脊髓血供的个体差异有关。

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引用本文的文献

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