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1
CCR2-V64I polymorphism is associated with increased risk of cervical cancer but not with HPV infection or pre-cancerous lesions in African women.CCR2-V64I 多态性与宫颈癌风险增加相关，但与非洲妇女的 HPV 感染或癌前病变无关。
BMC Cancer. 2010 Jun 10;10:278. doi: 10.1186/1471-2407-10-278.
2
Fas and FasL gene polymorphisms are not associated with cervical cancer but differ among Black and Mixed-ancestry South Africans.Fas和FasL基因多态性与宫颈癌无关，但在南非黑人和混血人种中存在差异。
BMC Res Notes. 2009 Nov 26;2:238. doi: 10.1186/1756-0500-2-238.
3
Association of two CASP8 polymorphisms with breast cancer risk: a meta-analysis.两个 CASP8 多态性与乳腺癌风险的关联：荟萃分析。
Breast Cancer Res Treat. 2010 Feb;120(1):229-34. doi: 10.1007/s10549-009-0471-5. Epub 2009 Jul 24.
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Death receptor signal transducers: nodes of coordination in immune signaling networks.死亡受体信号转导分子：免疫信号网络中的协调节点
Nat Immunol. 2009 Apr;10(4):348-55. doi: 10.1038/ni.1714. Epub 2009 Mar 19.
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Candidate gene identification approach: progress and challenges.候选基因鉴定方法：进展与挑战。
Int J Biol Sci. 2007 Oct 25;3(7):420-7. doi: 10.7150/ijbs.3.420.
6
Predisposition to HPV16/18-related cervical cancer because of proline homozygosity at codon 72 of p53 among Indian women is influenced by HLA-B*07 and homozygosity of HLA-DQB1*03.印度女性中，由于p53基因第72位密码子的脯氨酸纯合性导致的HPV16/18相关宫颈癌易感性受HLA - B*07和HLA - DQB1*03纯合性影响。
Tissue Antigens. 2007 Oct;70(4):283-93. doi: 10.1111/j.1399-0039.2007.00894.x.
7
Major histocompatibility complex class II polymorphisms and risk of cervical cancer in Puerto Rican women.主要组织相容性复合体II类多态性与波多黎各女性患宫颈癌的风险
P R Health Sci J. 2007 Jun;26(2):97-101.
8
Variants of chemokine receptor 2 and interleukin 4 receptor, but not interleukin 10 or Fas ligand, increase risk of cervical cancer.趋化因子受体2和白细胞介素4受体的变体而非白细胞介素10或Fas配体，会增加患宫颈癌的风险。
Int J Cancer. 2007 Dec 1;121(11):2451-7. doi: 10.1002/ijc.22989.
9
The influence of chemokine receptor CCR2 genotypes in the route to cervical carcinogenesis.趋化因子受体CCR2基因分型在宫颈癌发生途径中的影响。
Gynecol Obstet Invest. 2007;64(4):208-12. doi: 10.1159/000106492. Epub 2007 Jul 30.
10
HLA polymorphisms and cervical human Papillomavirus infection in a cohort of Montreal University students.蒙特利尔大学生队列中的HLA多态性与宫颈人乳头瘤病毒感染
J Infect Dis. 2007 Jul 1;196(1):82-90. doi: 10.1086/518612. Epub 2007 May 29.

关于宿主对人乳头瘤病毒感染及进展为宫颈癌的遗传易感性的综合综述。

A comprehensive review on host genetic susceptibility to human papillomavirus infection and progression to cervical cancer.

作者信息

Chattopadhyay Koushik

机构信息

Nelson R. Mandela School of Medicine, University of KwaZulu-Natal, Durban, Republic of South Africa.

出版信息

Indian J Hum Genet. 2011 Sep;17(3):132-44. doi: 10.4103/0971-6866.92087.

DOI:10.4103/0971-6866.92087
PMID:22345983
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3276980/
Abstract

Cervical cancer is the second most common cancer in women worldwide. This is caused by oncogenic types of human papillomavirus (HPV) infection. Although large numbers of young sexually active women get HPV-infected, only a small fraction develop cervical cancer. This points to different co-factors for regression of HPV infection or progression to cervical cancer. Host genetic factors play an important role in the outcome of such complex or multifactor diseases such as cervical cancer and are also known to regulate the rate of disease progression. The aim of this review is to compile the advances in the field of host genetics of cervical cancer. MEDLINE database was searched using the terms, 'HPV', 'cervical', 'CIN', 'polymorphism(s)', 'cervical'+ the name of the gene and 'HPV'+ the name of the gene. This review focuses on the major host genes reported to affect the progression to cervical cancer in HPV infected individuals.

摘要

宫颈癌是全球女性中第二常见的癌症。这是由致癌型人乳头瘤病毒（HPV）感染引起的。尽管大量年轻的性活跃女性会感染HPV，但只有一小部分会发展为宫颈癌。这表明HPV感染消退或发展为宫颈癌存在不同的共同因素。宿主遗传因素在宫颈癌等复杂或多因素疾病的发生发展中起着重要作用，并且已知其可调节疾病进展速度。本综述的目的是汇总宫颈癌宿主遗传学领域的进展。使用“HPV”、“宫颈”、“CIN”、“多态性”、“宫颈” + 基因名称以及“HPV” + 基因名称等检索词在MEDLINE数据库中进行了检索。本综述重点关注据报道会影响HPV感染个体发展为宫颈癌的主要宿主基因。