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植物中除草剂 2,4-D 的毒性机制和细胞反应的研究进展。

Insights into the toxicity mechanism of and cell response to the herbicide 2,4-D in plants.

机构信息

Departamento de Bioquímica, Biología Celular y Molecular de Plantas; Estación Experimental del Zaidín, CSIC, Granada, Spain.

出版信息

Plant Signal Behav. 2012 Mar;7(3):425-7. doi: 10.4161/psb.19124. Epub 2012 Mar 1.

Abstract

Although structurally similar to the natural plant hormone indol-3- acetic acid, auxin herbicides were developed for purposes other than growth, and have been successfully used in agriculture for the last 60 years. Concerted efforts are being made to understand and decipher the precise mechanism of action of IAA and synthetic auxins. Innumerable results need to be interconnected to resolve the puzzle of auxin biology and action mode of auxin herbicides. To date, different breakthroughs are providing more insights into the process of plant-herbicide interactions. Here we highlight some of the latest findings on how the 2,4-dichlorophenoxyacetic acid damages susceptible broadleaf plants, emphasizing the role of ROS as a downstream component of the auxin signal transduction under herbicide treatment.

摘要

尽管结构上与天然植物激素吲哚-3-乙酸相似,但生长素除草剂的开发目的并非生长,而是在过去 60 年中成功应用于农业。人们正在努力理解和破译 IAA 和合成生长素的确切作用机制。需要将无数的结果相互关联,以解决生长素生物学和生长素除草剂作用模式的难题。迄今为止,不同的突破为植物-除草剂相互作用的过程提供了更多的见解。在这里,我们重点介绍一些关于 2,4-二氯苯氧乙酸如何损害敏感阔叶植物的最新发现,强调 ROS 作为除草剂处理下生长素信号转导的下游组成部分的作用。

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