Nebraska Biomechanics Core Facility, University of Nebraska at Omaha, Omaha, NE 68182-0216, United States.
Gait Posture. 2012 Jul;36(3):506-9. doi: 10.1016/j.gaitpost.2012.05.004. Epub 2012 Jun 5.
Previous studies have shown major deficits in gait for individuals with peripheral arterial disease before and after the onset of pain. However, these studies did not have subjects ambulate at similar velocities and potential exists that the differences in joint powers may have been due to differences in walking velocity. The purpose of this study was to examine the joint moments and powers of peripheral arterial disease limbs for subjects walking at similar self-selected walking velocities as healthy controls prior to onset of any symptoms. Results revealed peripheral arterial disease patients have reduced peak hip power absorption in midstance (p=0.017), reduced peak knee power absorption in early and late stance (p=0.037 and p=0.020 respectively), and reduced peak ankle power generation in late stance (p=0.021). This study reveals that the gait of patients with peripheral arterial disease walking prior to the onset of any leg symptoms is characterized by failure of specific and identifiable muscle groups needed to perform normal walking and that these gait deficits are independent of reduced gait velocity.
先前的研究表明，在疼痛发作之前和之后，外周动脉疾病患者的步态存在明显缺陷。然而，这些研究并没有让受试者以相似的速度行走，并且关节功率的差异可能是由于行走速度的差异造成的。本研究的目的是检查外周动脉疾病肢体的关节力矩和功率，这些受试者在出现任何症状之前以类似于健康对照者的自我选择行走速度行走。结果显示，外周动脉疾病患者在中步时的髋关节峰值功率吸收减少（p=0.017），在早步和晚步时的膝关节峰值功率吸收减少（p=0.037 和 p=0.020），以及在晚步时的踝关节峰值功率生成减少（p=0.021）。这项研究表明，在出现任何腿部症状之前行走的外周动脉疾病患者的步态表现为无法进行正常行走所需的特定和可识别的肌肉群的功能失效，而这些步态缺陷与行走速度的降低无关。
