Institute of Molecular Biosciences, University of Graz, 8010 Graz, Austria.
Mol Cell. 2012 Jun 8;46(5):552-4. doi: 10.1016/j.molcel.2012.05.032.
In this issue of Molecular Cell, Dwyer et al. (2012) characterize a RecA-dependent and ClpXP-regulated pathway that controls the acquisition of several apoptotic markers upon bactericidal treatment of prokaryotes, placing the hypothetical origin of apoptosis further downstream in evolution.
在本期《分子细胞》中，Dwyer 等人（2012）描述了一个依赖 RecA 和 ClpXP 的调控途径，该途径控制了原核生物在杀菌处理后获得几种凋亡标记物，从而将凋亡的假设起源进一步推到了进化的下游。
