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人类外周血单核细胞表现出不均一的 CD52 表达水平,并对阿仑单抗介导的细胞溶解显示出不同的敏感性。

Human peripheral blood mononuclear cells exhibit heterogeneous CD52 expression levels and show differential sensitivity to alemtuzumab mediated cytolysis.

机构信息

Neuroimmunology Research, Genzyme, A Sanofi Company, Framingham, Massachusetts, United States of America.

出版信息

PLoS One. 2012;7(6):e39416. doi: 10.1371/journal.pone.0039416. Epub 2012 Jun 25.

Abstract

Alemtuzumab is a monoclonal antibody that targets cell surface CD52 and is effective in depleting lymphocytes by cytolytic effects in vivo. Although the cytolytic effects of alemtuzumab are dependent on the density of CD52 antigen on cells, there is scant information regarding the expression levels of CD52 on different cell types. In this study, CD52 expression was assessed on phenotypically distinct subsets of lymphoid and myeloid cells in peripheral blood mononuclear cells (PBMCs) from normal donors. Results demonstrate that subsets of PBMCs express differing levels of CD52. Quantitative analysis showed that memory B cells and myeloid dendritic cells (mDCs) display the highest number while natural killer (NK) cells, plasmacytoid dendritic cells (pDCs) and basophils have the lowest number of CD52 molecules per cell amongst lymphoid and myeloid cell populations respectively. Results of complement dependent cytolysis (CDC) studies indicated that alemtuzumab mediated profound cytolytic effects on B and T cells with minimal effect on NK cells, basophils and pDCs, correlating with the density of CD52 on these cells. Interestingly, despite high CD52 levels, mDCs and monocytes were less susceptible to alemtuzumab-mediated CDC indicating that antigen density alone does not define susceptibility. Additional studies indicated that higher expression levels of complement inhibitory proteins (CIPs) on these cells partially contributes to their resistance to alemtuzumab mediated CDC. These results indicate that alemtuzumab is most effective in depleting cells of the adaptive immune system while leaving innate immune cells relatively intact.

摘要

阿仑单抗是一种靶向细胞表面 CD52 的单克隆抗体,通过体内的细胞溶解作用有效地耗竭淋巴细胞。虽然阿仑单抗的细胞溶解作用依赖于细胞上 CD52 抗原的密度,但关于不同细胞类型上 CD52 的表达水平的信息很少。在这项研究中,评估了来自正常供体的外周血单个核细胞(PBMC)中不同表型的淋巴样和髓样细胞亚群上的 CD52 表达。结果表明,PBMC 亚群表达不同水平的 CD52。定量分析显示,记忆 B 细胞和髓样树突状细胞(mDC)显示最高数量,而自然杀伤(NK)细胞、浆细胞样树突状细胞(pDC)和嗜碱性粒细胞在淋巴样和髓样细胞群体中每个细胞的 CD52 分子数量最低。补体依赖性细胞溶解(CDC)研究的结果表明,阿仑单抗对 B 和 T 细胞具有深远的细胞溶解作用,对 NK 细胞、嗜碱性粒细胞和 pDC 的作用最小,这与这些细胞上 CD52 的密度相关。有趣的是,尽管 CD52 水平较高,但 mDC 和单核细胞对阿仑单抗介导的 CDC 的敏感性较低,表明抗原密度本身并不能定义敏感性。进一步的研究表明,这些细胞上补体抑制蛋白(CIP)的高表达水平部分导致它们对阿仑单抗介导的 CDC 的抗性。这些结果表明,阿仑单抗在耗竭适应性免疫系统的细胞方面最为有效,而使固有免疫细胞相对完整。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8ca5/3382607/50773831fb3c/pone.0039416.g001.jpg

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