Department of Orthopaedic Surgery, William Beaumont Hospital, 3535 W 13 Mile Road #744, Royal Oak, MI 48073, USA.
Clin Orthop Relat Res. 2012 Dec;470(12):3383-9. doi: 10.1007/s11999-012-2453-x.
Cam-type, pincer, and mixed femoroacetabular impingement (FAI) are accepted causes of labral and acetabular rim injury; however, the abnormal contact stresses associated with motion may damage other areas of the hip. Although cartilage damage to the femoral head has been reported previously in athletes, FAI-associated focal parafoveal chondral defects differ from previously reported lesions and represent a rare manifestation of the complex pathomechanics associated with FAI.
QUESTIONS/PURPOSES: We describe the clinical, radiographic, and surgical characteristics of a rare focal anterolateral parafoveal femoral chondral defect associated with FAI.
We retrospectively reviewed 10 patients with symptomatic FAI diagnosed with this unique focal defect confirmed at the time of surgical dislocation. Patients presented with hip pain, clinical findings of FAI, and, frequently, with an identifiable lesion on MRI arthrography. The minimum clinical followup was 12 months (mean, 29 months; range, 12-72 months).
The consistent characteristics of these lesions associated with FAI differ from previously reported femoral chondral damage reported after hip dislocation or lateral impact in that there was no discrete injury such as a fall or dislocation/subluxation, no associated traumatic femoral lesion, and all were localized to the posterosuperior femoral head. Eight of 10 were diagnosed preoperatively using MR arthrography.
Despite radiographic similarities to findings of osteoarthritis and osteonecrosis, these FAI-associated femoral chondral defects were amenable to surgical reconstruction using first- or second-generation cartilage repair techniques during surgical treatment of impingement. The etiology of these lesions may be related to complex intraarticular forces generated by FAI-associated transient hip subluxation or forceful nonconcentric motion.
Level IV, therapeutic study. See Guidelines for Authors for a complete description of levels of evidence.
凸轮型、钳夹型和混合型股骨髋臼撞击症(FAI)是导致盂唇和髋臼缘损伤的公认原因;然而,与运动相关的异常接触应力可能会损伤髋关节的其他区域。虽然先前有报道称运动员的股骨头软骨损伤,但 FAI 相关的局灶性前外侧窝状软骨缺损与先前报道的病变不同,代表了与 FAI 相关的复杂病理力学的罕见表现。
问题/目的:我们描述了与 FAI 相关的罕见局灶性前外侧窝状股骨软骨缺损的临床、影像学和手术特征。
我们回顾性分析了 10 例经手术脱位时确诊为这种独特局灶性缺陷的症状性 FAI 患者。患者表现为髋关节疼痛、FAI 的临床发现,并且经常在 MRI 关节造影上发现有可识别的病变。最低临床随访时间为 12 个月(平均 29 个月;范围,12-72 个月)。
这些与 FAI 相关的病变的一致特征与髋关节脱位或外侧撞击后报告的先前报道的股骨软骨损伤不同,没有明确的损伤,如跌倒或脱位/半脱位、无相关的创伤性股骨病变,并且均局限于股骨头后上侧。10 例中有 8 例术前通过 MRI 关节造影术诊断。
尽管这些 FAI 相关的股骨软骨缺损在影像学上与骨关节炎和股骨头坏死的表现相似,但在治疗撞击症时,使用第一代或第二代软骨修复技术进行手术重建是可行的。这些病变的病因可能与 FAI 相关的髋关节一过性半脱位或强力非同心运动产生的复杂关节内力有关。
IV 级,治疗研究。有关证据水平的完整描述,请参见作者指南。