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自然杀伤细胞系统中的靶效应细胞相互作用。IV. 环核苷酸的调节作用

Target-effector interaction in the natural killer cell system. IV. Modulation by cyclic nucleotides.

作者信息

Roder J C, Klein M

出版信息

J Immunol. 1979 Dec;123(6):2785-90.

PMID:227959
Abstract

Dibutyryl cAMP (dB-cAMP) and the cAMP elevating agents, prostaglandin E1, theophylline, and histamine markedly suppressed NK cytolytic function in a dose- and rate-dependent manner. The inhibition was rapidly induced and persisted in the presence of the drugs. Separate pretreatment of targets and highly purified NK cells, isolated by a target binding and velocity sedimentation technique, revealed that PGE1 and dB-cAMP acted at the level of the effector cell in a short-term cytolytic assay. In contrast to the inhibitory effects of cAMP elevating agents, dB-cGMP and carbamylcholine caused a small but significant acceleration in the rate of lysis and could compete with inhibitory doses of dB-cAMP to reduce the level of suppression thereby suggesting that the cAMP-cGMP ratio might be important in NK-mediated lysis. Insulin had no effect on NK activity, whereas T cell-mediated cytolysis was augmented by insulin and cGMP if the effector cells were taken early after alloimmunization but not later. Neither cAMP- nor cGMP-elevating agents affected the frequency of NK-target cell conjugates. These results are compatible with the hypothesis that cyclic nucleotides may be involved in triggering the lytic event within NK cells.

摘要

二丁酰环磷腺苷(dB - cAMP)以及环磷腺苷升高剂、前列腺素E1、茶碱和组胺,均以剂量和速率依赖性方式显著抑制自然杀伤细胞(NK)的细胞溶解功能。这种抑制作用诱导迅速,且在药物存在的情况下持续存在。通过靶细胞结合和速度沉降技术分离的靶细胞和高度纯化的NK细胞的单独预处理显示,在短期细胞溶解试验中,前列腺素E1和dB - cAMP作用于效应细胞水平。与环磷腺苷升高剂的抑制作用相反,dB - cGMP和氨甲酰胆碱使溶解速率有小幅但显著的加快,并且可以与抑制剂量的dB - cAMP竞争以降低抑制水平,从而表明环磷腺苷 - 环磷鸟苷比值可能在NK介导的溶解中起重要作用。胰岛素对NK活性无影响,而如果在同种异体免疫后早期而非晚期获取效应细胞,胰岛素和环磷鸟苷会增强T细胞介导的细胞溶解。环磷腺苷升高剂和环磷鸟苷升高剂均不影响NK - 靶细胞结合物的频率。这些结果与环核苷酸可能参与触发NK细胞内溶解事件的假说相符。

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