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Scientific letter: Ac-SDKP (N-acetyl-seryl-aspartyl-lysyl-proline) and Galectin-3 levels in tuberculous pericardial effusion: implications for pathogenesis and prevention of pericardial constriction.

作者信息

Ntsekhe Mpiko, Matthews Kerryn, Wolske Janine, Badri Motasim, Wilkinson Katalin A, Wilkinson Robert J, Sturrock Edward D, Mayosi Bongani M

出版信息

Heart. 2012 Sep;98(17):1326-8. doi: 10.1136/heartjnl-2012-302196. Epub 2012 Jul 26.

DOI:10.1136/heartjnl-2012-302196
PMID:22842991
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4180888/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c69b/4180888/3d19b5ad3a1e/emss-60443-f0002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c69b/4180888/be024f231552/emss-60443-f0001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c69b/4180888/3d19b5ad3a1e/emss-60443-f0002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c69b/4180888/be024f231552/emss-60443-f0001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c69b/4180888/3d19b5ad3a1e/emss-60443-f0002.jpg

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1
Scientific letter: Ac-SDKP (N-acetyl-seryl-aspartyl-lysyl-proline) and Galectin-3 levels in tuberculous pericardial effusion: implications for pathogenesis and prevention of pericardial constriction.科研信：结核性心包积液中Ac-SDKP（N-乙酰基-丝氨酰-天冬氨酰-赖氨酰-脯氨酸）和半乳糖凝集素-3水平：对心包缩窄发病机制及预防的意义
Heart. 2012 Sep;98(17):1326-8. doi: 10.1136/heartjnl-2012-302196. Epub 2012 Jul 26.
2
The Effects of Angiotensin Converting Enzyme Inhibitors (ACE-I) on Human N-Acetyl-Seryl-Aspartyl-Lysyl-Proline (Ac-SDKP) Levels: A Systematic Review and Meta-Analysis.血管紧张素转换酶抑制剂（ACE-I）对人N-乙酰丝氨酰-天冬氨酰-赖氨酰-脯氨酸（Ac-SDKP）水平的影响：一项系统评价和荟萃分析
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N-acetyl-seryl-aspartyl-lysyl-proline prevents cardiac remodeling and dysfunction induced by galectin-3, a mammalian adhesion/growth-regulatory lectin.N-乙酰丝氨酰-天冬氨酰-赖氨酰-脯氨酸可预防由半乳糖凝集素-3（一种哺乳动物黏附/生长调节凝集素）诱导的心脏重塑和功能障碍。
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Investigating the antifibrotic potential of N-acetyl seryl-aspartyl-lysyl-proline sequence peptides.研究 N-乙酰丝氨酰-天冬氨酰-赖氨酰-脯氨酸序列肽的抗纤维化潜力。
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Front Cardiovasc Med. 2024 Mar 18;11:1329767. doi: 10.3389/fcvm.2024.1329767. eCollection 2024.
2
Synthesis and Identification of a Novel Peptide, Ac-SDK (Biotin) Proline, That Can Elicit Anti-Fibrosis Effects in Rats Suffering from Silicosis.合成并鉴定一种新型肽，Ac-SDK（生物素）脯氨酸，可在矽肺大鼠中产生抗纤维化作用。
Drug Des Devel Ther. 2020 Oct 19;14:4315-4326. doi: 10.2147/DDDT.S262716. eCollection 2020.
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Galectin-3 Levels in Patients with Chronic Constrictive Pericarditis.慢性缩窄性心包炎患者的半乳糖凝集素-3 水平。
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N-Acetyl-Seryl-Aspartyl-Lysyl-Proline Mitigates Experimental Colitis Through Inhibition of Intestinal Mucosal Inflammatory Responses MEK-ERK Signaling.N-乙酰丝氨酰-天冬氨酰-赖氨酰-脯氨酸通过抑制肠道黏膜炎症反应的MEK-ERK信号通路减轻实验性结肠炎。
Front Pharmacol. 2020 May 6;11:593. doi: 10.3389/fphar.2020.00593. eCollection 2020.
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本文引用的文献

1
Risk of constrictive pericarditis after acute pericarditis.急性心包炎后缩窄性心包炎的风险。
Circulation. 2011 Sep 13;124(11):1270-5. doi: 10.1161/CIRCULATIONAHA.111.018580. Epub 2011 Aug 15.
2
Prevention of myocardial fibrosis by N-acetyl-seryl-aspartyl-lysyl-proline in diabetic rats.N-乙酰丝氨酰-天门冬酰-赖氨酰-脯氨酸预防糖尿病大鼠心肌纤维化。
Clin Sci (Lond). 2009 Oct 26;118(3):211-20. doi: 10.1042/cs20090234.
3
N-acetyl-seryl-aspartyl-lysyl-proline prevents cardiac remodeling and dysfunction induced by galectin-3, a mammalian adhesion/growth-regulatory lectin.N-乙酰丝氨酰-天冬氨酰-赖氨酰-脯氨酸可预防由半乳糖凝集素-3（一种哺乳动物黏附/生长调节凝集素）诱导的心脏重塑和功能障碍。
Am J Physiol Heart Circ Physiol. 2009 Feb;296(2):H404-12. doi: 10.1152/ajpheart.00747.2008. Epub 2008 Dec 19.
4
Galectin-3 expression and secretion links macrophages to the promotion of renal fibrosis.半乳糖凝集素-3的表达与分泌将巨噬细胞与肾纤维化的进展联系起来。
Am J Pathol. 2008 Feb;172(2):288-98. doi: 10.2353/ajpath.2008.070726. Epub 2008 Jan 17.
5
Decreased endogenous levels of Ac-SDKP promote organ fibrosis.内源性Ac-SDKP水平降低会促进器官纤维化。
Hypertension. 2007 Jul;50(1):130-6. doi: 10.1161/HYPERTENSIONAHA.106.084103. Epub 2007 Apr 30.