• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

相似文献

1
Tumor microenvironment regulates epithelial-mesenchymal transitions in metastasis.肿瘤微环境在转移过程中调节上皮-间质转化。
Expert Rev Anticancer Ther. 2012 Jul;12(7):857-9. doi: 10.1586/era.12.69.
2
Interplay of Immunometabolism and Epithelial-Mesenchymal Transition in the Tumor Microenvironment.免疫代谢与肿瘤微环境中上皮-间充质转化的相互作用。
Int J Mol Sci. 2021 Sep 13;22(18):9878. doi: 10.3390/ijms22189878.
3
The tumor microenvironment: An irreplaceable element of tumor budding and epithelial-mesenchymal transition-mediated cancer metastasis.肿瘤微环境：肿瘤芽生和上皮-间质转化介导的癌症转移中不可替代的要素。
Cell Adh Migr. 2016 Jul 3;10(4):434-46. doi: 10.1080/19336918.2015.1129481. Epub 2016 Jan 8.
4
Whom to blame for metastasis, the epithelial-mesenchymal transition or the tumor microenvironment?转移应归咎于上皮-间质转化还是肿瘤微环境？
Cancer Lett. 2016 Sep 28;380(1):359-68. doi: 10.1016/j.canlet.2015.12.033. Epub 2016 Jan 11.
5
Revisiting epithelial-mesenchymal transition in cancer metastasis: the connection between epithelial plasticity and stemness.重新审视癌症转移中的上皮-间质转化：上皮可塑性与干性之间的联系。
Mol Oncol. 2017 Jul;11(7):792-804. doi: 10.1002/1878-0261.12096. Epub 2017 Jun 26.
6
Tumor microenvironment and noncoding RNAs as co-drivers of epithelial-mesenchymal transition and cancer metastasis.肿瘤微环境与非编码RNA作为上皮-间质转化和癌症转移的共同驱动因素
Dev Dyn. 2018 Mar;247(3):405-431. doi: 10.1002/dvdy.24548. Epub 2017 Sep 18.
7
The tumor ecosystem regulates the roads for invasion and metastasis.肿瘤生态系统调控着侵袭和转移的途径。
Clin Res Hepatol Gastroenterol. 2011 Nov;35(11):714-9. doi: 10.1016/j.clinre.2011.05.003. Epub 2011 Jun 14.
8
Emerging role of tumor microenvironment derived exosomes in therapeutic resistance and metastasis through epithelial-to-mesenchymal transition.肿瘤微环境衍生的外泌体通过上皮间质转化在治疗抵抗和转移中的新作用。
Life Sci. 2021 Sep 1;280:119750. doi: 10.1016/j.lfs.2021.119750. Epub 2021 Jun 23.
9
EMT and MET: necessary or permissive for metastasis?上皮-间质转化和间质-上皮转化：转移的必要条件还是允许条件？
Mol Oncol. 2017 Jul;11(7):755-769. doi: 10.1002/1878-0261.12083. Epub 2017 Jun 12.
10
Concepts of metastasis in flux: the stromal progression model.转移概念的转变：基质演进模型。
Semin Cancer Biol. 2012 Jun;22(3):174-86. doi: 10.1016/j.semcancer.2012.02.007. Epub 2012 Feb 21.

引用本文的文献

1
Epithelial-mesenchymal transition (EMT) and its role in acquired epidermal growth factor receptor-tyrosine kinase inhibitor (EGFR-TKI) chemoresistance in non-small cell lung cancer (NSCLC).上皮-间质转化（EMT）及其在非小细胞肺癌（NSCLC）获得性表皮生长因子受体-酪氨酸激酶抑制剂（EGFR-TKI）化疗耐药中的作用。
Cancer Pathog Ther. 2024 Jul 6;3(3):215-225. doi: 10.1016/j.cpt.2024.07.001. eCollection 2025 May.
2
A Metastatic Cancer Expression Generator (MetGen): A Generative Contrastive Learning Framework for Metastatic Cancer Generation.一种转移性癌症表达生成器（MetGen）：用于生成转移性癌症的生成性对比学习框架。
Cancers (Basel). 2024 Apr 25;16(9):1653. doi: 10.3390/cancers16091653.
3
Paradigm shift in the pathogenesis and treatment of oral cancer and other cancers focused on the oralome and antimicrobial-based therapeutics.口腔癌和其他癌症的发病机制和治疗范式的转变，聚焦于口腔组学和基于抗菌的治疗方法。
Periodontol 2000. 2021 Oct;87(1):76-93. doi: 10.1111/prd.12388.
4
Epithelial to Mesenchymal Transition History: From Embryonic Development to Cancers.上皮间质转化历史：从胚胎发育到癌症。
Biomolecules. 2021 May 22;11(6):782. doi: 10.3390/biom11060782.
5
Cancer drug resistance induced by EMT: novel therapeutic strategies.上皮间质转化诱导的癌症耐药性：新的治疗策略。
Arch Toxicol. 2021 Jul;95(7):2279-2297. doi: 10.1007/s00204-021-03063-7. Epub 2021 May 18.
6
Jiedu Sangen Decoction Inhibits Migration and Invasion of Colon Cancer SW480 Cells via Suppressing Epithelial Mesenchymal Transition.解毒三参汤通过抑制上皮-间质转化抑制结肠癌SW480细胞的迁移和侵袭。
Evid Based Complement Alternat Med. 2018 Sep 25;2018:1495768. doi: 10.1155/2018/1495768. eCollection 2018.
7
Preclinical Efficacy and Safety Assessment of Artemisinin-Chemotherapeutic Agent Conjugates for Ovarian Cancer.青蒿素-化疗药物偶联物用于卵巢癌的临床前疗效与安全性评估
EBioMedicine. 2016 Dec;14:44-54. doi: 10.1016/j.ebiom.2016.11.026. Epub 2016 Nov 23.
8
A weighty problem: metabolic perturbations and the obesity-cancer link.一个重大问题：代谢紊乱与肥胖-癌症关联
Horm Mol Biol Clin Investig. 2015 Aug;23(2):47-57. doi: 10.1515/hmbci-2015-0022.
9
Crosstalk between cancer cells and blood endothelial and lymphatic endothelial cells in tumour and organ microenvironment.肿瘤及器官微环境中癌细胞与血液内皮细胞和淋巴管内皮细胞之间的相互作用。
Expert Rev Mol Med. 2015 Jan 30;17:e3. doi: 10.1017/erm.2015.2.

本文引用的文献

1
Myeloid progenitor cells in the premetastatic lung promote metastases by inducing mesenchymal to epithelial transition.前转移肺部的髓系祖细胞通过诱导上皮间质转化促进转移。
Cancer Res. 2012 Mar 15;72(6):1384-94. doi: 10.1158/0008-5472.CAN-11-2905. Epub 2012 Jan 26.
2
Augmented autocrine bone morphogenic protein (BMP) 7 signaling increases the metastatic potential of mouse breast cancer cells.增强的自分泌骨形态发生蛋白 7 信号增加了小鼠乳腺癌细胞的转移潜力。
Clin Exp Metastasis. 2012 Apr;29(4):327-38. doi: 10.1007/s10585-012-9453-9. Epub 2012 Jan 25.
3
Intratumoral macrophages contribute to epithelial-mesenchymal transition in solid tumors.肿瘤内巨噬细胞促进实体瘤中的上皮间质转化。
BMC Cancer. 2012 Jan 24;12:35. doi: 10.1186/1471-2407-12-35.
4
EMT and dissemination precede pancreatic tumor formation. EMT 和播散先于胰腺肿瘤形成。
Cell. 2012 Jan 20;148(1-2):349-61. doi: 10.1016/j.cell.2011.11.025.
5
Whole genome sequencing of matched primary and metastatic acral melanomas.肢端黑色素瘤原发灶与转移灶的全基因组测序。
Genome Res. 2012 Feb;22(2):196-207. doi: 10.1101/gr.125591.111. Epub 2011 Dec 19.
6
Bone morphogenetic protein 7 in dormancy and metastasis of prostate cancer stem-like cells in bone.骨形态发生蛋白 7 在前列腺癌干细胞样细胞骨内休眠和转移中的作用。
J Exp Med. 2011 Dec 19;208(13):2641-55. doi: 10.1084/jem.20110840. Epub 2011 Nov 28.
7
Mesenchymal transition and dissemination of cancer cells is driven by myeloid-derived suppressor cells infiltrating the primary tumor.间质转化和癌细胞的扩散是由浸润原发性肿瘤的髓系来源抑制细胞驱动的。
PLoS Biol. 2011 Sep;9(9):e1001162. doi: 10.1371/journal.pbio.1001162. Epub 2011 Sep 27.
8
Prostate tumor cell plasticity: a consequence of the microenvironment.前列腺肿瘤细胞的可塑性：源于微环境。
Adv Exp Med Biol. 2011;720:81-90. doi: 10.1007/978-1-4614-0254-1_7.
9
Direct targeting of Sec23a by miR-200s influences cancer cell secretome and promotes metastatic colonization.miR-200s 通过直接靶向 Sec23a 影响癌细胞分泌组并促进转移定植。
Nat Med. 2011 Aug 7;17(9):1101-8. doi: 10.1038/nm.2401.
10
Phenotypic plasticity and epithelial-mesenchymal transitions in cancer and normal stem cells?癌症和正常干细胞中的表型可塑性和上皮-间充质转化？
Int J Cancer. 2011 Nov 15;129(10):2310-4. doi: 10.1002/ijc.26311. Epub 2011 Aug 29.

Tumor microenvironment regulates epithelial-mesenchymal transitions in metastasis.

作者信息

Gao Dingcheng, Mittal Vivek

出版信息

Expert Rev Anticancer Ther. 2012 Jul;12(7):857-9. doi: 10.1586/era.12.69.

DOI:10.1586/era.12.69
PMID:22845398
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4656980/
Abstract
摘要