Wing R R, Blair E H, Epstein L H, McDermott M D
Department of Psychiatry, University of Pittsburgh School of Medicine, PA 15213.
Health Psychol. 1990;9(6):693-700. doi: 10.1037//0278-6133.9.6.693.
Acute psychological stress has been found to impair the handling of a glucose load in normal-weight individuals and to produce a delay in gastrointestinal transit time. The present study utilized the same paradigm to compare 10 obese and 10 normal-weight subjects. All subjects participated in two counterbalanced sessions (stress and nonstress). At each session, subjects were fed a carbohydrate load, and glucose was measured 0, 30, 60, 90, and 120 min after the load. On nonstress days, subjects relaxed after the drink; on stress days, subjects participated in 30 min of competitive tasks. The stressor significantly delayed the glucose response in the normal-weight subjects but did not affect the glucose response in obese subjects. This finding may have implications for differences between obese and normal-weight individuals in stress-induced eating, possibly explaining the decreased consumption of normal-weight individuals during stress.
研究发现，急性心理压力会损害正常体重个体对葡萄糖负荷的处理能力，并导致胃肠运输时间延迟。本研究采用相同的范式对10名肥胖受试者和10名正常体重受试者进行了比较。所有受试者均参加了两个相互平衡的实验环节（压力和无压力）。在每个环节中，给受试者喂食碳水化合物负荷，并在负荷后0、30、60、90和120分钟测量血糖。在无压力日，受试者在饮用后放松；在压力日，受试者参加30分钟的竞争性任务。压力源显著延迟了正常体重受试者的血糖反应，但对肥胖受试者的血糖反应没有影响。这一发现可能对肥胖个体和正常体重个体在压力诱导进食方面的差异具有启示意义，可能解释了正常体重个体在压力期间摄入量减少的原因。
