Comment on: Turban et al. Optimal elevation of β-cell 11β-hydroxysteroid dehydrogenase type 1 is a compensatory mechanism that prevents high-fat diet-induced β-cell failure. Diabetes 2012;61:642-652.
作者信息
Liu Jun-Li, Srikant Coimbatore B, Chowdhury Subrata
出版信息
Diabetes. 2012 Sep;61(9):e13; author reply e14. doi: 10.2337/db12-0556.
Abstract
摘要
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2
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本文引用的文献
1
Optimal elevation of β-cell 11β-hydroxysteroid dehydrogenase type 1 is a compensatory mechanism that prevents high-fat diet-induced β-cell failure.最佳升高β细胞 11β-羟类固醇脱氢酶 1 型是一种代偿机制，可防止高脂肪饮食诱导的β细胞衰竭。
Diabetes. 2012 Mar;61(3):642-52. doi: 10.2337/db11-1054. Epub 2012 Feb 7.
2
Pancreatic-specific inactivation of IGF-I gene causes enlarged pancreatic islets and significant resistance to diabetes.胰岛素样生长因子-I基因在胰腺中的特异性失活会导致胰岛增大，并对糖尿病产生显著抗性。
Diabetes. 2004 Dec;53(12):3131-41. doi: 10.2337/diabetes.53.12.3131.
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