人类白细胞抗原同种免疫的预防：白细胞去除术和紫外线B照射的作用
Prevention of HLA alloimmunization: role of leukocyte depletion and UV-B irradiation.
作者信息
Snyder E L
机构信息
Department of Laboratory Medicine, Yale University School of Medicine, New Haven, Connecticut.
出版信息
Yale J Biol Med. 1990 Sep-Oct;63(5):419-27.
Abstract
HLA alloimmunization is a major cause of the platelet refractory state. The stimulus for HLA alloimmunization is believed to derive from incompatibility between the recipient's lymphocytes and the passenger donor lymphocytes contained in transfused red cells or platelet concentrates. Two techniques to prevent post-transfusion HLA alloimmunization include filtration, which physically removes the donor lymphocytes, and UV-B irradiation, which renders the donor leukocytes biologically inactive. The role of these two techniques in the prevention of HLA alloimmunization is the focus of this review.
摘要
人类白细胞抗原（HLA）同种免疫是血小板输注无效状态的主要原因。HLA同种免疫的刺激因素据信源于受者淋巴细胞与输注的红细胞或血小板浓缩物中所含供者过客淋巴细胞之间的不相容性。预防输血后HLA同种免疫的两种技术包括过滤，即从物理上清除供者淋巴细胞，以及紫外线B（UV-B）照射，使供者白细胞失去生物学活性。这两种技术在预防HLA同种免疫中的作用是本综述的重点。
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