Bischoff C, Schönle P W
Abteilung Klinische Neurophysiologie, Zentrum Neurologische Medizin der Universität Göttingen.
Dtsch Med Wochenschr. 1990 Jan 19;115(3):98-101. doi: 10.1055/s-2008-1064977.
Following a viral infection of the upper respiratory tract a 63-year-old woman developed a postinfectious myopathy with nonprogressive muscle weakness and diffuse muscle pain. Electromyography revealed typical changes of a myopathy with low-amplitude, splintered voluntary potentials and prematurely dense interference pattern, without additional abnormal spontaneous activity as a pointer to an acute inflammatory process. Muscle biopsy showed moderate focal atrophic and inflammatory changes but no perivascular inflammation. The clinical and biochemical tests were noncontributory and there was no evidence for collagen disease. No cortisone was given because of the minor degree of the postinfectious myopathy. The symptoms completely regressed within ten months.
一名63岁女性在上呼吸道病毒感染后出现感染后肌病，伴有非进行性肌无力和弥漫性肌肉疼痛。肌电图显示肌病的典型变化，即低振幅、破碎的自发电位和过早密集的干扰模式，无额外异常自发电活动提示急性炎症过程。肌肉活检显示中度局灶性萎缩和炎症改变，但无血管周围炎症。临床和生化检查无诊断意义，也无胶原病证据。由于感染后肌病程度较轻，未给予皮质类固醇治疗。症状在十个月内完全消退。
