在犊牛败血性沙门氏菌病期间，无法证明循环肿瘤坏死因子的诱导。
Induction of circulating tumor necrosis factor cannot be demonstrated during septicemic salmonellosis in calves.
作者信息
Peel J E, Voirol M J, Kolly C, Gobet D, Martinod S
机构信息
Centre de Recherches Agricoles, Ciba-Geigy S.A., St. Aubin, Switzerland.
出版信息
Infect Immun. 1990 Feb;58(2):439-42. doi: 10.1128/iai.58.2.439-442.1990.
Abstract
The concentration of tumor necrosis factor in the circulation of calves, which were infected with Salmonella typhimurium and exhibited septicemia as indicated by clinical signs and blood culture, was measured with a radioimmunoassay. These levels were compared with those in calves before infection and in other calves that had received an intravenous dose of gram-negative endotoxin. The tumor necrosis factor levels measured in samples taken during septicemia were not different from those in samples from infected nonsepticemic calves or samples from calves before infection. In contrast, the levels of tumor necrosis factor rose rapidly in calves after treatment with endotoxin by intravenous injection.
摘要
对感染鼠伤寒沙门氏菌并出现临床症状和血培养显示为败血症的犊牛循环中的肿瘤坏死因子浓度，采用放射免疫测定法进行测量。将这些水平与感染前犊牛以及接受静脉注射革兰氏阴性内毒素的其他犊牛的水平进行比较。败血症期间采集的样本中测得的肿瘤坏死因子水平与感染但未患败血症的犊牛样本或感染前犊牛样本中的水平并无差异。相比之下，静脉注射内毒素治疗后，犊牛体内的肿瘤坏死因子水平迅速升高。
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