Trosch R M, Ransom B R
Department of Neurology, Yale University School of Medicine, New Haven, CT 06510.
Neurology. 1990 Feb;40(2):376-7. doi: 10.1212/wnl.40.2.376.
A 66-year-old man suffered bilateral subdural hematomas progressing to central herniation, despite repeated surgical evacuations. This eventually resolved, leaving him with a severe parkinsonian syndrome that was responsive to levodopa. MRI and CT showed midbrain compression from central herniation, and a follow-up MRI revealed thinning of the pars compacta. The clinical and radiologic evidence suggested that midbrain compression from central herniation was the probable cause of parkinsonism in this patient.
一名66岁男性尽管多次接受手术清除血肿，但仍出现双侧硬膜下血肿并进展为中央疝。最终病情得到缓解，但留下了对左旋多巴有反应的严重帕金森综合征。MRI和CT显示中央疝导致中脑受压，后续MRI显示黑质致密部变薄。临床和影像学证据表明，中央疝导致的中脑受压可能是该患者帕金森症的病因。
