Evans J R, McIntosh J P, McIntosh H J, Mitchell P E
Department of Biochemical Medicine, Stracathro Hospital, Brechin, Scotland, U.K.
Clin Chem. 1990 Feb;36(2):322-5.
Calculation of osmolal gap in plasma and urine samples from patients after acute myocardial infarction was carried out to see whether changes in these variables support the concept of a "sick-cell" mechanism being responsible for the hyponatremia associated with acute myocardial infarction. Some supportive evidence was found on the first day after admission but, overall, the evidence was not convincing.
对急性心肌梗死后患者的血浆和尿液样本进行了渗透压间隙计算，以观察这些变量的变化是否支持“病态细胞”机制导致急性心肌梗死相关低钠血症这一概念。入院后第一天发现了一些支持性证据，但总体而言，证据并不令人信服。
