Katayama T, Iwamoto K, Ochi S, Honda Y, Shigematsu K
Department of Cardiology, Nagasaki Citizens Hospital, Japan.
Angiology. 1990 Jan;41(1):76-81. doi: 10.1177/000331979004100112.
A fifty-five-year-old man developed intractable heart failure four weeks after upper respiratory infection. His central venous pressure was as high as 300 mmH2O. Two-dimensional echocardiogram revealed no muscular hypertrophy of the ventricles, no pericardial thickness, and no pericardial effusion. On catheterization, hemodynamic data were compatible with restrictive cardiomyopathy. Transvenous endomyocardial biopsy of the left ventricle was performed. Histologic examination strongly suggested that cardiomyopathy developed after acute myocarditis.
一名55岁男性在上呼吸道感染四周后出现顽固性心力衰竭。他的中心静脉压高达300 mmH2O。二维超声心动图显示心室无肌肉肥厚，心包无增厚，无心包积液。心导管检查显示血流动力学数据符合限制性心肌病。对左心室进行了经静脉心内膜心肌活检。组织学检查强烈提示心肌病是在急性心肌炎后发生的。
