Effect of inhaled endotoxin on regional particle deposition in patients with mild asthma.
作者信息
Bennett William D, Herbst Margaret, Zeman Kirby L, Wu Jihong, Hernandez Michelle L, Peden David B
出版信息
J Allergy Clin Immunol. 2013 Mar;131(3):912-3. doi: 10.1016/j.jaci.2012.09.010. Epub 2012 Oct 25.
Abstract
摘要
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Atopic asthmatic patients have reduced airway inflammatory cell recruitment after inhaled endotoxin challenge compared with healthy volunteers.特应性哮喘患者在吸入内毒素后，与健康志愿者相比，气道炎症细胞募集减少。
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Effect of inhaled dust mite allergen on regional particle deposition and mucociliary clearance in allergic asthmatics.吸入尘螨过敏原对过敏性哮喘患者区域性颗粒沉积和黏液纤毛清除的影响。
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LPS-stimulated MUC5AC production involves Rac1-dependent MMP-9 secretion and activation in NCI-H292 cells.脂多糖刺激的黏蛋白5AC产生涉及NCI-H292细胞中Rac1依赖性基质金属蛋白酶-9的分泌和激活。
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Inflammatory response of the airway to inhaled endotoxin correlates with body mass index in atopic patients with asthma but not in normal volunteers.在患有哮喘的特应性患者中，气道对吸入内毒素的炎症反应与体重指数相关，但在正常志愿者中并非如此。
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Low-dose airborne endotoxin exposure enhances bronchial responsiveness to inhaled allergen in atopic asthmatics.
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