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Nuclear factor-κB in pancreatitis: Jack-of-all-trades, but which one is more important?
Gastroenterology. 2013 Jan;144(1):26-9. doi: 10.1053/j.gastro.2012.11.016. Epub 2012 Nov 16.
2
Deletion of IκBα activates RelA to reduce acute pancreatitis in mice through up-regulation of Spi2A.
Gastroenterology. 2013 Jan;144(1):192-201. doi: 10.1053/j.gastro.2012.09.058. Epub 2012 Oct 3.
3
Activation of nuclear factor-κB in acinar cells increases the severity of pancreatitis in mice.
Gastroenterology. 2013 Jan;144(1):202-10. doi: 10.1053/j.gastro.2012.09.059. Epub 2012 Oct 3.
4
Karyopherin Alpha 2 Promotes the Inflammatory Response in Rat Pancreatic Acinar Cells Via Facilitating NF-κB Activation.
Dig Dis Sci. 2016 Mar;61(3):747-57. doi: 10.1007/s10620-015-3948-6. Epub 2015 Nov 2.
6
NIK and IKKbeta interdependence in NF-kappaB signalling--flux analysis of regulation through metabolites.
Biosystems. 2010 Feb;99(2):140-9. doi: 10.1016/j.biosystems.2009.10.009. Epub 2009 Nov 10.
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Suppression of interleukin-6 production in macrophages by furonaphthoquinone NFD-37.
Int Immunopharmacol. 2006 Jun;6(6):916-23. doi: 10.1016/j.intimp.2006.01.006. Epub 2006 Feb 3.
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ERβ regulation of NF-kB activation in prostate cancer is mediated by HIF-1.
Oncotarget. 2015 Nov 24;6(37):40247-54. doi: 10.18632/oncotarget.5377.
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BCL3 Reduces the Sterile Inflammatory Response in Pancreatic and Biliary Tissues.
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Neuroadaptations in human chronic alcoholics: dysregulation of the NF-kappaB system.
PLoS One. 2007 Sep 26;2(9):e930. doi: 10.1371/journal.pone.0000930.

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Predictive Value of Serum , and Gene Expression in Acute Pancreatitis Outcomes.
Diagnostics (Basel). 2025 Aug 26;15(17):2160. doi: 10.3390/diagnostics15172160.
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Oxidative stress, DAMPs, and immune cells in acute pancreatitis: molecular mechanisms and therapeutic prospects.
Front Immunol. 2025 Aug 20;16:1608618. doi: 10.3389/fimmu.2025.1608618. eCollection 2025.
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Trypsin in pancreatitis: The culprit, a mediator, or epiphenomenon?
World J Gastroenterol. 2024 Nov 7;30(41):4417-4438. doi: 10.3748/wjg.v30.i41.4417.
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NF-κB and Pancreatic Cancer; Chapter and Verse.
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PPAR-Alpha Agonist Fenofibrate Combined with Octreotide Acetate in the Treatment of Acute Hyperlipidemia Pancreatitis.
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Apigenin Decreases Acinar Cell Damage in Pancreatitis.
Pancreas. 2019 May/Jun;48(5):711-718. doi: 10.1097/MPA.0000000000001310.
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Acute Pancreatitis: A Multifaceted Set of Organelle and Cellular Interactions.
Gastroenterology. 2019 May;156(7):1941-1950. doi: 10.1053/j.gastro.2018.11.082. Epub 2019 Jan 18.
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Acinar cell injury induced by inadequate unfolded protein response in acute pancreatitis.
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本文引用的文献

1
Deletion of IκBα activates RelA to reduce acute pancreatitis in mice through up-regulation of Spi2A.
Gastroenterology. 2013 Jan;144(1):192-201. doi: 10.1053/j.gastro.2012.09.058. Epub 2012 Oct 3.
2
Activation of nuclear factor-κB in acinar cells increases the severity of pancreatitis in mice.
Gastroenterology. 2013 Jan;144(1):202-10. doi: 10.1053/j.gastro.2012.09.059. Epub 2012 Oct 3.
4
NF-κB and the link between inflammation and cancer.
Immunol Rev. 2012 Mar;246(1):379-400. doi: 10.1111/j.1600-065X.2012.01099.x.
5
Role of NF-κB in epithelial biology.
Immunol Rev. 2012 Mar;246(1):346-58. doi: 10.1111/j.1600-065X.2012.01109.x.
6
Dimer-specific regulatory mechanisms within the NF-κB family of transcription factors.
Immunol Rev. 2012 Mar;246(1):193-204. doi: 10.1111/j.1600-065X.2011.01091.x.
7
It takes two to tango: IκBs, the multifunctional partners of NF-κB.
Immunol Rev. 2012 Mar;246(1):59-76. doi: 10.1111/j.1600-065X.2012.01102.x.
8
NF-κB, the first quarter-century: remarkable progress and outstanding questions.
Genes Dev. 2012 Feb 1;26(3):203-34. doi: 10.1101/gad.183434.111.
9
Inflammasomes in health and disease.
Nature. 2012 Jan 18;481(7381):278-86. doi: 10.1038/nature10759.
10
Intra-acinar trypsinogen activation mediates early stages of pancreatic injury but not inflammation in mice with acute pancreatitis.
Gastroenterology. 2011 Dec;141(6):2210-2217.e2. doi: 10.1053/j.gastro.2011.08.033. Epub 2011 Aug 27.

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