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FLC-4 细胞,一种人肝癌细胞,呈球形细胞形态,通过整合 mRNA- microRNA 相互作用,增强了肝细胞特异性功能并抑制了肿瘤表型。

Spherical cell shape of FLC-4 cell, a human hepatoma cell, enhances hepatocyte-specific function and suppresses tumor phenotype through the integration of mRNA-microRNA interaction.

机构信息

Laboratory of Nutritional Biochemistry, Department of Applied Molecular Biosciences, Nagoya University , Nagoya 464-8601 , Japan.

出版信息

Biol Open. 2012 Oct 15;1(10):958-64. doi: 10.1242/bio.20121438. Epub 2012 Aug 8.

DOI:10.1242/bio.20121438
PMID:23213373
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3507180/
Abstract

The induction mechanism of HNF-4α by spherical cell shape in human hepatoma cells, FLC-4, was investigated. To get insight into the induction mechanism of HNF-4α in three-dimensional FLC-4 cells, mRNA microarray analysis was performed. The gene expression related to drug metabolism and nuclear receptors, such as LXRα, was elevated in spherical FLC-4 cells. We found the first time that the expressions of genes related to malignancy of hepatoma cells, such as HIF-1α, c-Myc and VEGFC, were downregulated by spherical cell shape. Network analysis revealed that HNF-4α would elicit both the enhancement of hepatocyte-specific gene expression and suppression of malignancy. Since HNF-4α gene expression was known to be regulated by microRNA, we inferred that spherical cell shape would induce HNF-4α gene expression through microRNA. To investigate the possibility of such a mechanism, mRNA-microRNA interactions were examined using microRNA microarray and bioinformatics analysis. The level of miR-24, a microRNA targeting HNF-4α, was reduced in spherical FLC-4 cells. On the other hand, spherical cell shape-induced miR-194 and miR-320c would directly downregulate SLC7A5 and E2F1 gene expression, respectively, which are both related to malignancy. Our study suggested that spherical cell shape would induce HNF-4α gene expression and consequent enhancement hepatocyte-specific functions. Spherical cell shape itself would suppress malignancy in FLC-4 cells through microRNA, such as miR-194 and miR-320c.

摘要

本文研究了人肝癌细胞 FLC-4 中球形细胞形状诱导 HNF-4α 的机制。为了深入了解三维 FLC-4 细胞中 HNF-4α 的诱导机制,我们进行了 mRNA 微阵列分析。球形 FLC-4 细胞中与药物代谢和核受体相关的基因,如 LXRα,表达上调。我们首次发现,与肝癌细胞恶性相关的基因,如 HIF-1α、c-Myc 和 VEGFC 的表达,通过球形细胞形状下调。网络分析表明,HNF-4α 会引发肝细胞特异性基因表达的增强和恶性的抑制。由于 HNF-4α 基因表达受 microRNA 调控,我们推断球形细胞形状可能通过 microRNA 诱导 HNF-4α 基因表达。为了研究这种机制的可能性,我们使用 microRNA 微阵列和生物信息学分析检查了 mRNA-microRNA 相互作用。靶向 HNF-4α 的 microRNA miR-24 的水平在球形 FLC-4 细胞中降低。另一方面,球形细胞形状诱导的 miR-194 和 miR-320c 分别直接下调 SLC7A5 和 E2F1 基因表达,这两者都与恶性有关。我们的研究表明,球形细胞形状会诱导 HNF-4α 基因表达,并随之增强肝细胞特异性功能。球形细胞形状本身通过 microRNA,如 miR-194 和 miR-320c,抑制 FLC-4 细胞中的恶性。

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HIF-1α coordinates lymphangiogenesis during wound healing and in response to inflammation.HIF-1α 在伤口愈合和炎症反应过程中协调淋巴管生成。
FASEB J. 2012 Mar;26(3):1027-39. doi: 10.1096/fj.11-195321. Epub 2011 Nov 8.
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A novel human hepatoma cell line, FLC-4, exhibits highly enhanced liver differentiation functions through the three-dimensional cell shape.
一种新型的人肝癌细胞系 FLC-4,通过三维细胞形态表现出高度增强的肝分化功能。
J Cell Physiol. 2012 Jul;227(7):2898-906. doi: 10.1002/jcp.23033.
4
MiR-192 inhibits nucleotide excision repair by targeting ERCC3 and ERCC4 in HepG2.2.15 cells.miR-192 通过靶向 HepG2.2.15 细胞中的 ERCC3 和 ERCC4 抑制核苷酸切除修复。
Biochem Biophys Res Commun. 2011 Jul 8;410(3):440-5. doi: 10.1016/j.bbrc.2011.05.153. Epub 2011 Jun 6.
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Expression profile analysis of the inflammatory response regulated by hepatocyte nuclear factor 4α.肝细胞核因子 4α 调控的炎症反应表达谱分析。
BMC Genomics. 2011 Feb 25;12:128. doi: 10.1186/1471-2164-12-128.
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HIF-1α confers aggressive malignant traits on human tumor cells independent of its canonical transcriptional function.缺氧诱导因子-1α(HIF-1α)通过非经典转录功能赋予人类肿瘤细胞侵袭性恶性特征。
Cancer Res. 2011 Feb 15;71(4):1244-52. doi: 10.1158/0008-5472.CAN-10-2360. Epub 2011 Feb 8.
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