Zidar Janez, Bäckman Eva, Bengtsson Ann, Henriksson K G
Inst. of Clinical Neurophysiology, University Medical Centre, LjubljanaYugoslavia Dept. of Clinical Neurophysiology, University Hospital, LinköpingSweden Division of Rheumatology, Dept. of Internal Medicine, University Hospital, LinköpingSweden Neuromuscular Unit, University Hospital, LinköpingSweden.
Pain. 1990 Mar;40(3):249-254. doi: 10.1016/0304-3959(90)91122-Y.
Examinations were performed in 22 female fibromyalgic patients and in 9 healthy controls. The biceps brachii, trapezius, and tibial anterior muscles were examined electromyographically. The changes found were minor and non-specific. This implies that there is no important loss of motor units and no conspicuous muscle fiber degeneration in fibromyalgia. Our investigation also failed to demonstrate any electrically detectable muscle activity in muscles where the patients during the examination reported pain (paraspinal, trapezius and tibial anterior muscles). This means that muscle tension cannot be a prominent pathogenetic factor in fibromyalgia and that factors other than muscle tension are responsible for maintaining the pain in fibromyalgia.
对22名女性纤维肌痛患者和9名健康对照者进行了检查。对肱二头肌、斜方肌和胫前肌进行了肌电图检查。发现的变化轻微且不具特异性。这意味着纤维肌痛中不存在运动单位的重要丧失，也没有明显的肌纤维变性。我们的研究还未能在患者在检查期间报告疼痛的肌肉（椎旁肌、斜方肌和胫前肌）中检测到任何电可检测的肌肉活动。这意味着肌肉紧张不可能是纤维肌痛的主要致病因素，并且除肌肉紧张之外的其他因素是导致纤维肌痛疼痛持续的原因。
