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MUC5B: a good target for future therapy in pulmonary fibrosis?

作者信息

Mahida Rahul, Turner Alice M

出版信息

Thorax. 2013 May;68(5):401. doi: 10.1136/thoraxjnl-2012-202957. Epub 2013 Jan 17.

DOI:10.1136/thoraxjnl-2012-202957
PMID:23329274
Abstract
摘要

相似文献

1
MUC5B: a good target for future therapy in pulmonary fibrosis?MUC5B:肺纤维化未来治疗的良好靶点?
Thorax. 2013 May;68(5):401. doi: 10.1136/thoraxjnl-2012-202957. Epub 2013 Jan 17.
2
Mucin 5B promoter polymorphism is associated with idiopathic pulmonary fibrosis but not with development of lung fibrosis in systemic sclerosis or sarcoidosis.黏蛋白 5B 启动子多态性与特发性肺纤维化相关,但与系统性硬化症或结节病肺纤维化的发展无关。
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3
The pulmonary fibrosis-associated MUC5B promoter polymorphism does not influence the development of interstitial pneumonia in systemic sclerosis.肺纤维化相关的 MUC5B 启动子多态性不影响系统性硬化症间质性肺炎的发生。
Chest. 2012 Dec;142(6):1584-1588. doi: 10.1378/chest.12-0110.
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MUC5B promoter polymorphism and pulmonary fibrosis.MUC5B启动子多态性与肺纤维化
N Engl J Med. 2011 Jul 14;365(2):178; author reply 178-9. doi: 10.1056/NEJMc1105707.
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The Possible Pathogenesis of Idiopathic Pulmonary Fibrosis considering .考虑......的特发性肺纤维化的可能发病机制。
Biomed Res Int. 2019 Jun 11;2019:9712464. doi: 10.1155/2019/9712464. eCollection 2019.
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MUC5B Promoter Polymorphism and Survival in Indian Patients With Idiopathic Pulmonary Fibrosis.MUC5B启动子多态性与印度特发性肺纤维化患者的生存情况
Chest. 2022 Oct;162(4):824-827. doi: 10.1016/j.chest.2022.04.001. Epub 2022 Apr 8.
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Prognostic role of MUC5B rs35705950 genotype in patients with idiopathic pulmonary fibrosis (IPF) on antifibrotic treatment.MUC5B rs35705950 基因型在接受抗纤维化治疗的特发性肺纤维化 (IPF) 患者中的预后作用。
Respir Res. 2021 Apr 1;22(1):98. doi: 10.1186/s12931-021-01694-z.
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MUC5B promoter polymorphism and pulmonary fibrosis.MUC5B启动子多态性与肺纤维化
N Engl J Med. 2011 Jul 14;365(2):178; author reply 178-9. doi: 10.1056/NEJMc1105707.
9
MUC5B and Idiopathic Pulmonary Fibrosis.黏蛋白5B与特发性肺纤维化
Ann Am Thorac Soc. 2015 Nov;12 Suppl 2(Suppl 2):S193-9. doi: 10.1513/AnnalsATS.201503-110AW.
10
Interaction between the promoter polymorphism and mucin expression: is there a difference according to ILD subtype?启动子多态性与黏蛋白表达的相互作用:根据间质性肺病亚型是否存在差异?
Thorax. 2020 Oct;75(10):901-903. doi: 10.1136/thoraxjnl-2020-214579. Epub 2020 Jun 24.

引用本文的文献

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Trends and hotspots in pulmonary fibrosis biomarker research: a bibliometric analysis.肺纤维化生物标志物研究的趋势与热点:一项文献计量分析
Front Med (Lausanne). 2025 Apr 25;12:1541364. doi: 10.3389/fmed.2025.1541364. eCollection 2025.
2
Context-specific eQTLs provide deeper insight into causal genes underlying shared genetic architecture of COVID-19 and idiopathic pulmonary fibrosis.特定背景下的eQTL为深入了解新冠病毒肺炎和特发性肺纤维化共同遗传结构背后的因果基因提供了线索。
HGG Adv. 2025 Apr 10;6(2):100410. doi: 10.1016/j.xhgg.2025.100410. Epub 2025 Jan 27.
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Context-specific eQTLs reveal causal genes underlying shared genetic architecture of critically ill COVID-19 and idiopathic pulmonary fibrosis.
特定背景下的eQTL揭示了重症COVID-19和特发性肺纤维化共同遗传结构背后的因果基因。
medRxiv. 2024 Jul 14:2024.07.13.24310305. doi: 10.1101/2024.07.13.24310305.
4
MUC5B promoter polymorphisms and risk of coal workers' pneumoconiosis in a Chinese population.中国人群中MUC5B启动子多态性与煤工尘肺风险
Mol Biol Rep. 2014 Jul;41(7):4171-6. doi: 10.1007/s11033-014-3100-2. Epub 2014 Jun 13.
5
The MUC5B variant is associated with idiopathic pulmonary fibrosis but not with systemic sclerosis interstitial lung disease in the European Caucasian population.MUC5B 变异与特发性肺纤维化相关,但与欧洲白种人群的系统性硬化症间质性肺病无关。
PLoS One. 2013 Aug 5;8(8):e70621. doi: 10.1371/journal.pone.0070621. Print 2013.