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本文引用的文献
1
Targeting the UPR transcription factor XBP1 protects against Huntington's disease through the regulation of FoxO1 and autophagy.
Hum Mol Genet. 2012 May 15;21(10):2245-62. doi: 10.1093/hmg/dds040. Epub 2012 Feb 14.
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The unfolded protein response: controlling cell fate decisions under ER stress and beyond.
Nat Rev Mol Cell Biol. 2012 Jan 18;13(2):89-102. doi: 10.1038/nrm3270.
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BAX inhibitor-1 regulates autophagy by controlling the IRE1α branch of the unfolded protein response.
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JNK regulates FoxO-dependent autophagy in neurons.
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Regulation of glucose homeostasis through a XBP-1-FoxO1 interaction.
Nat Med. 2011 Mar;17(3):356-65. doi: 10.1038/nm.2293. Epub 2011 Feb 13.
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Translational pausing ensures membrane targeting and cytoplasmic splicing of XBP1u mRNA.
Science. 2011 Feb 4;331(6017):586-9. doi: 10.1126/science.1197142. Epub 2011 Jan 13.
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Cytosolic FoxO1 is essential for the induction of autophagy and tumour suppressor activity.
Nat Cell Biol. 2010 Jul;12(7):665-75. doi: 10.1038/ncb2069. Epub 2010 Jun 13.
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Direct proteasome binding and subsequent degradation of unspliced XBP-1 prevent its intracellular aggregation.
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XBP-1 deficiency in the nervous system protects against amyotrophic lateral sclerosis by increasing autophagy.
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Cotranslational targeting of XBP1 protein to the membrane promotes cytoplasmic splicing of its own mRNA.
Mol Cell. 2009 Apr 24;34(2):191-200. doi: 10.1016/j.molcel.2009.02.033.
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