Lisachenko G V, Ivanova N A
Biull Eksp Biol Med. 1990 Jan;109(1):16-8.
Experiments were performed on 43 dogs with injured (infarction) and intact myocardium typical of the cardiac output changes with initial transitory increasing, following decreasing and tendency for restitution in 24 h after resuscitation were established. Similar transferences of fluid in body volumes with initial entrance of fluid in cells, which bring the development of hypovolemia and following return in extracellular volume were shown. More pronounced depression of contractile function of the heart in combination with retarded of restitution plasma volume and delay of fluid in interstitium of dogs with myocardial infarction accompany change for the worse of restitution and survival.
对43只患有损伤(梗死)和完整心肌的狗进行了实验,确定了心输出量变化的典型情况,即复苏后最初短暂增加,随后下降,并在24小时内有恢复趋势。研究显示,身体容积中的液体转移情况类似,最初液体进入细胞,导致血容量减少,随后细胞外液量恢复。心肌梗死犬的心脏收缩功能明显降低,同时血浆容量恢复延迟,液体在间质中潴留,这伴随着恢复情况和生存率的恶化。