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本文引用的文献

1
Behavioral stress accelerates prostate cancer development in mice.行为应激加速小鼠前列腺癌的发展。
J Clin Invest. 2013 Feb;123(2):874-86. doi: 10.1172/JCI63324. Epub 2013 Jan 25.
2
The influence of glucocorticoid signaling on tumor progression.糖皮质激素信号对肿瘤进展的影响。
Brain Behav Immun. 2013 Mar;30 Suppl(0):S26-31. doi: 10.1016/j.bbi.2012.10.022. Epub 2012 Nov 16.
3
Social influences on clinical outcomes of patients with ovarian cancer.社会因素对卵巢癌患者临床结局的影响。
J Clin Oncol. 2012 Aug 10;30(23):2885-90. doi: 10.1200/JCO.2011.39.4411. Epub 2012 Jul 16.
4
Neuroendocrine influences on cancer progression.神经内分泌对癌症进展的影响。
Brain Behav Immun. 2013 Mar;30 Suppl(Suppl):S19-25. doi: 10.1016/j.bbi.2012.06.005. Epub 2012 Jun 21.
5
Social isolation is associated with elevated tumor norepinephrine in ovarian carcinoma patients.社会隔离与卵巢癌患者肿瘤去甲肾上腺素水平升高有关。
Brain Behav Immun. 2011 Feb;25(2):250-5. doi: 10.1016/j.bbi.2010.10.012. Epub 2010 Oct 16.
6
Adrenergic modulation of focal adhesion kinase protects human ovarian cancer cells from anoikis.肾上腺素能调节黏着斑激酶可保护人卵巢癌细胞免于失巢凋亡。
J Clin Invest. 2010 May;120(5):1515-23. doi: 10.1172/JCI40802. Epub 2010 Apr 12.
7
Norepinephrine upregulates VEGF, IL-8, and IL-6 expression in human melanoma tumor cell lines: implications for stress-related enhancement of tumor progression.去甲肾上腺素上调人黑色素瘤肿瘤细胞系中VEGF、IL-8和IL-6的表达：对与应激相关的肿瘤进展增强的影响。
Brain Behav Immun. 2009 Feb;23(2):267-75. doi: 10.1016/j.bbi.2008.10.005. Epub 2008 Oct 21.
8
The evolving biology and treatment of prostate cancer.前列腺癌不断发展的生物学特性与治疗方法
J Clin Invest. 2007 Sep;117(9):2351-61. doi: 10.1172/JCI31791.
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Epinephrine protects cancer cells from apoptosis via activation of cAMP-dependent protein kinase and BAD phosphorylation.肾上腺素通过激活环磷酸腺苷（cAMP）依赖性蛋白激酶和BAD磷酸化来保护癌细胞免于凋亡。
J Biol Chem. 2007 May 11;282(19):14094-100. doi: 10.1074/jbc.M611370200. Epub 2007 Mar 12.
10
Chronic stress promotes tumor growth and angiogenesis in a mouse model of ovarian carcinoma.在卵巢癌小鼠模型中，慢性应激促进肿瘤生长和血管生成。
Nat Med. 2006 Aug;12(8):939-44. doi: 10.1038/nm1447. Epub 2006 Jul 23.

为什么压力对癌症患者有害。

Why stress is BAD for cancer patients.

机构信息

Department of Gynecologic Oncology and Reproductive Medicine, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

出版信息

J Clin Invest. 2013 Feb;123(2):558-60. doi: 10.1172/JCI67887. Epub 2013 Jan 25.

DOI:10.1172/JCI67887
PMID:23348736
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3561841/
Abstract

Behavioral stress is known to promote tumor progression in experimental models, but the role of behavioral stress in cancer initiation is less clear. In this issue, Hassan et al. focus on the signaling and biological effects induced by stress hormones that lead to tumor cell evasion from apoptosis, resulting in prostate cancer progression.

摘要

行为应激已知可促进实验模型中的肿瘤进展，但行为应激在癌症起始中的作用尚不清楚。在本期杂志中，Hassan 等人关注应激激素诱导的信号转导和生物学效应，这些效应导致肿瘤细胞逃避细胞凋亡，从而促进前列腺癌进展。