Kinsella F, Kyle P, Stansfield A
Department of Ophthalmology, Southern General Hospital, Glasgow.
Br J Ophthalmol. 1990 Apr;74(4):239-41. doi: 10.1136/bjo.74.4.239.
We report a case of bilateral complete failure of adduction following bilateral translid antralethmoidal orbital decompression. We believe the probable mechanism is neuropraxia (temporary dysfunction) of the third cranial nerves' supply to the medial recti, owing to these nerves' occupying an anatomically abnormal position. Partial recovery of adduction occurred over the ensuing six months.
我们报告一例双侧经鼻泪囊筛窦眶减压术后双侧内收完全性障碍的病例。我们认为可能的机制是第三对颅神经向内直肌供血出现神经失用（暂时性功能障碍），原因是这些神经处于解剖学异常位置。在随后的六个月内，内收功能部分恢复。
