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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Clin Sci Mol Med. 1975 Jan;48(1):33-7. doi: 10.1042/cs0480033.

The distribution of ammonia between extra-and intra-cellular compartments of the brain was evaluated in anaesthetized rats exposed to sustained hyperammonaemia and superimposed acute hypo- or hyper-capnia. 2. It is concluded that the concentration gradient for ammonia between arterial plasma and cerebro-spinal fluid (CSF) cannot be explained by pH-dependent non-ionic diffusion of ammonia. A continuous uptake by the brain tissue of ammonia from the CSF is postulated and possible mechanisms are discussed.

在暴露于持续性高氨血症并叠加急性低碳酸血症或高碳酸血症的麻醉大鼠中，评估了大脑细胞外和细胞内区室之间氨的分布情况。2. 得出的结论是，动脉血浆和脑脊液（CSF）之间氨的浓度梯度不能用氨的pH依赖性非离子扩散来解释。推测脑组织持续从脑脊液中摄取氨，并讨论了可能的机制。

Clin Sci Mol Med. 1975 Jan;48(1):33-7. doi: 10.1042/cs0480033.

The distribution of ammonia between extra-and intra-cellular compartments of the brain was evaluated in anaesthetized rats exposed to sustained hyperammonaemia and superimposed acute hypo- or hyper-capnia. 2. It is concluded that the concentration gradient for ammonia between arterial plasma and cerebro-spinal fluid (CSF) cannot be explained by pH-dependent non-ionic diffusion of ammonia. A continuous uptake by the brain tissue of ammonia from the CSF is postulated and possible mechanisms are discussed.

在暴露于持续性高氨血症并叠加急性低碳酸血症或高碳酸血症的麻醉大鼠中，评估了大脑细胞外和细胞内区室之间氨的分布情况。2. 得出的结论是，动脉血浆和脑脊液（CSF）之间氨的浓度梯度不能用氨的pH依赖性非离子扩散来解释。推测脑组织持续从脑脊液中摄取氨，并讨论了可能的机制。