Yaqub B A, al-Deeb S M, Daif A K, Sharif H S, Shamena A R, al-Jaberi M, Obeid T, Panayiotopoulos C C
Division of Neurology, King Khalid University Hospital, King Saud University, Riyadh, Saudi Arabia.
J Neurol Sci. 1990 Apr;96(1):29-40. doi: 10.1016/0022-510x(90)90054-q.
We describe 6 patients with ophthalmoplegia, ataxia and normal or exaggerated deep tendon reflexes. All had been preceded by a febrile illness and had a full recovery without sequelae. The brainstem auditory evoked potentials showed a localised lesion in the upper brainstem while the pattern shift visual evoked potentials were normal and did not show any additional silent lesions. CSF IgG oligoclonal bands were not detected in any of the patients. MRI in 2 patients showed a confluent high intensity lesion in the upper mesencephalon and thalamus involving white and gray matter. Follow-up ranged from 6 to 24 months and showed no relapse.
我们描述了6例患有眼肌麻痹、共济失调且深部腱反射正常或亢进的患者。所有患者之前均有发热性疾病，且均完全康复，无后遗症。脑干听觉诱发电位显示上脑干有局限性病变，而图形视觉诱发电位正常，未显示任何其他隐匿性病变。所有患者脑脊液中均未检测到IgG寡克隆带。2例患者的MRI显示中脑上部和丘脑有融合性高强度病变，累及白质和灰质。随访时间为6至24个月，均未复发。
