Stojan M, Slavícek J
Kardiologická laborator a II. interní klinika fakulty vseobecného lékarství Univerzity Karlovy, Praha.
Sb Lek. 1990 Apr;92(4):110-7.
On biological model of current due to damage with the suction electrode on the frog heart atrium in situ was given the survey of epicardial, intracellular and membraneous leads. The following data were found out: 1. Monophase epicardial recording is gradually starting from underpressure values -13.3 kPa to -40.0 kPa in the suction electrode when it is already constant; 2. In the course of sucking the originating monophase recording in lead with the suction electrode is spontaneously disappearing after a longer period (6-10 hrs) and changing into original multiphase one; 3. Intracellular potential up to 0.5 mm round suction electrode is lowered and it thus corresponds to monophase deformation in unipolar extracellular leads in this particular region; 4. After the original fall of the intracellular potential under the suction electrode higher underpressure causes the increase of this potential. Under local loss of membraneous tension the intracellular electrode reaches the branch of demarcation current inbetween the internal and external sides of sound membranes as well as the extracellular electrode.
对用吸力电极损伤蛙原位心脏心房时的电流生物学模型进行了心外膜、细胞内和膜导联的研究。发现以下数据:1. 单相心外膜记录在吸力电极压力已恒定的情况下,从负压值-13.3千帕逐渐开始至-40.0千帕;2. 在吸力电极导联中,在较长时间(6 - 10小时)后,最初产生的单相记录会自发消失并转变为原来的多相记录;3. 在吸力电极周围0.5毫米范围内的细胞内电位降低,因此与该特定区域单极细胞外导联中的单相变形相对应;4. 在吸力电极下细胞内电位最初下降后,更高的负压会导致该电位升高。在局部膜张力丧失的情况下,细胞内电极会到达健全膜内外侧之间的分界电流分支以及细胞外电极处。