Stojan M, Vacek Z, Slavícek J
Sb Lek. 1989 Oct;91(10):289-93.
In biological model experiments in frog heart morphological substrate was observed in current extinction due to damage under suction electrode. After short-term suction discharge monophase recording disappeared without demonstrable myocyte ultrastructure changes by means of reparation ad integrum. Monophase recording spontaneously disappeared in distinct changes of monocyte structure with enlarged spaces in between myofibrils, with breakdown of cristae in mitochondria and with dilatation of tubules and cisternae of the endoplasmic reticulum. This tissue disintegration leads to demarcation and forming of new cellular surfaces on the border with the healthy tissue so that the damaged area turns into a mere electric conductor.
在青蛙心脏的生物模型实验中,观察到在吸力电极作用下因损伤导致电流消失时的形态学基质。短期吸力放电后,单相记录消失,通过完全修复未显示出心肌细胞超微结构变化。单相记录在单核细胞结构发生明显变化时自发消失,肌原纤维之间的间隙增大,线粒体嵴破裂,内质网小管和池扩张。这种组织崩解导致在与健康组织的边界处形成新的细胞表面,从而使受损区域变成单纯的电导体。