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正常和发育不良髋关节的体内髋关节接触分布和骨撞击。

In vivo hip joint contact distribution and bony impingement in normal and dysplastic human hips.

机构信息

Department of Orthopaedic Surgery, Osaka Kosei Nenkin Hospital, 4-2-78, Fukushima, Fukushima-ku, Osaka, 553-0003, Japan.

出版信息

J Orthop Res. 2013 Oct;31(10):1611-9. doi: 10.1002/jor.22414. Epub 2013 Jun 26.

DOI:10.1002/jor.22414
PMID:23804572
Abstract

Our objectives were to clarify the 3D articular contact areas of the in vivo normal hip joint and acetabular dysplasia during specific positions using magnetic resonance imaging (MRI), voxel-based registration, and proximity mapping. Forty-two normal and 24 dysplastic hips were examined. MRI was performed at four positions: neutral; 45° flexion; 15° extension; and the Patrick position. Femur and pelvis bone models were reconstructed at the neutral position and superimposed over the images of each different position using voxel-based registration. The inferred cartilage contact and bony impingement were investigated using proximity mapping. The femoral head translated in the anterior or posteroinferior, anterosuperior, and posteroinferior direction from neutral to 45° flexion, 15° extension, and the Patrick position, respectively. Multiple regression analyses showed age, femoral head sphericity, and acetabular sphericity to be associated with higher hip instability. The present technique using subject-specific models revealed the in vivo hip joint contact area in a population of healthy individuals and dysplastic patients without radioactive exposure. These results can be used for analyzing disease progression in the dysplastic hip and pathogenesis of acetabular labral tear.

摘要

我们的目标是使用磁共振成像(MRI)、体素配准和临近映射技术,明确在特定体位下活体正常髋关节和髋臼发育不良的关节面接触区域。共检查了 42 个正常髋关节和 24 个发育不良髋关节。MRI 在四个体位下进行:中立位、45°屈曲位、15°伸展位和 Patrick 位。在中立位重建股骨和骨盆骨模型,并使用体素配准将其叠加到每个不同体位的图像上。使用临近映射研究推断的软骨接触和骨撞击。从中立位到 45°屈曲位、15°伸展位和 Patrick 位,股骨头分别在前或后下方、前上方和后下方方向平移。多元回归分析表明,年龄、股骨头球形度和髋臼球形度与更高的髋关节不稳定性相关。本研究采用个体化模型的技术,在健康个体和发育不良患者中,在无放射性暴露的情况下,揭示了活体髋关节的接触区域。这些结果可用于分析发育不良髋关节的疾病进展和髋臼唇撕裂的发病机制。

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