Vardi Y, Siroky M B
Department of Surgery, Veterans Affairs Medical Center, Boston, Massachusetts 02130.
J Urol. 1990 Sep;144(3):794-7. doi: 10.1016/s0022-5347(17)39594-0.
Using a previously developed canine model of erection, we measured pudendal artery and venous flow as well as pressure in the corpora cavernosa, glans penis, pudendal artery and vein. Penile erection was induced by pelvic nerve stimulation. Arterial flow increased by a factor of 2.5 within two seconds after the onset of stimulation. This was followed by a similar increase in venous flow about one second later. Spongiosal pressure began to rise about six seconds after nerve stimulation and always remained below cavernosal pressure. Cavernosal pressure began to rise after a latency of about 11 seconds. This latency period is accounted for by relaxation of the smooth muscle of the cavernosal sinusoids. Spongiosal pressure is maintained primarily by a high flow state through the glans penis while cavernosal pressure depends on a veno-occlusive mechanism.
我们使用先前建立的犬勃起模型，测量了阴部动脉和静脉血流以及海绵体、阴茎头、阴部动脉和静脉的压力。通过刺激盆腔神经诱导阴茎勃起。刺激开始后两秒内，动脉血流增加了2.5倍。大约一秒后，静脉血流出现类似增加。神经刺激后约六秒，海绵体压力开始上升，且始终低于海绵窦压力。海绵窦压力在约11秒的潜伏期后开始上升。该潜伏期是由海绵体窦状隙平滑肌的松弛所致。海绵体压力主要通过阴茎头的高血流状态维持，而海绵窦压力则依赖于静脉闭塞机制。
