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Amyloid β-peptide oligomers, ryanodine receptor-mediated Ca(2+) release, and Wnt-5a/Ca(2+) signaling: opposing roles in neuronal mitochondrial dynamics?淀粉样β肽寡聚体、兰尼碱受体介导的Ca(2+)释放和Wnt-5a/Ca(2+)信号传导：在神经元线粒体动力学中起相反作用？
Front Cell Neurosci. 2013 Jul 30;7:120. doi: 10.3389/fncel.2013.00120. eCollection 2013.
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Canonical Wnt signaling protects hippocampal neurons from Aβ oligomers: role of non-canonical Wnt-5a/Ca(2+) in mitochondrial dynamics.经典 Wnt 信号通路可保护海马神经元免受 Aβ寡聚体的侵害：非经典 Wnt-5a/Ca(2+)在线粒体动力学中的作用。
Front Cell Neurosci. 2013 Jun 25;7:97. doi: 10.3389/fncel.2013.00097. eCollection 2013.
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RyR2-Mediated Ca Release and Mitochondrial ROS Generation Partake in the Synaptic Dysfunction Caused by Amyloid β Peptide Oligomers.兰尼碱受体2介导的钙释放和线粒体活性氧生成参与淀粉样β肽寡聚体引起的突触功能障碍。
Front Mol Neurosci. 2017 Apr 25;10:115. doi: 10.3389/fnmol.2017.00115. eCollection 2017.
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Amyloid β-peptide oligomers stimulate RyR-mediated Ca2+ release inducing mitochondrial fragmentation in hippocampal neurons and prevent RyR-mediated dendritic spine remodeling produced by BDNF.淀粉样 β-肽寡聚体刺激 RyR 介导的 Ca2+ 释放，导致海马神经元线粒体碎片化，并阻止 BDNF 产生的 RyR 介导的树突棘重塑。
Antioxid Redox Signal. 2011 Apr 1;14(7):1209-23. doi: 10.1089/ars.2010.3287. Epub 2011 Jan 6.
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Amyloid β production is regulated by β2-adrenergic signaling-mediated post-translational modifications of the ryanodine receptor.淀粉样蛋白β的产生受β2-肾上腺素能信号介导的兰尼碱受体翻译后修饰调控。
J Biol Chem. 2017 Jun 16;292(24):10153-10168. doi: 10.1074/jbc.M116.743070. Epub 2017 May 5.
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Wnt-5a increases NO and modulates NMDA receptor in rat hippocampal neurons.Wnt-5a 增加了大鼠海马神经元中的 NO 并调节了 NMDA 受体。
Biochem Biophys Res Commun. 2014 Feb 7;444(2):189-94. doi: 10.1016/j.bbrc.2014.01.031. Epub 2014 Jan 16.
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The antioxidant N-acetylcysteine prevents the mitochondrial fragmentation induced by soluble amyloid-β peptide oligomers.抗氧化剂 N-乙酰半胱氨酸可预防可溶性淀粉样β肽寡聚物诱导的线粒体碎片化。
Neurodegener Dis. 2012;10(1-4):34-7. doi: 10.1159/000334901. Epub 2012 Jan 24.
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Wnt-5a occludes Abeta oligomer-induced depression of glutamatergic transmission in hippocampal neurons.Wnt-5a 阻断 Abeta 寡聚体诱导的海马神经元谷氨酸能传递抑制。
Mol Neurodegener. 2010 Jan 18;5:3. doi: 10.1186/1750-1326-5-3.
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Ca(2+) -dependent endoplasmic reticulum stress correlates with astrogliosis in oligomeric amyloid β-treated astrocytes and in a model of Alzheimer's disease.钙（Ca2+）依赖性内质网应激与寡聚淀粉样β处理的星形胶质细胞和阿尔茨海默病模型中的星形胶质细胞增生相关。
Aging Cell. 2013 Apr;12(2):292-302. doi: 10.1111/acel.12054. Epub 2013 Feb 28.
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Involvement of endoplasmic reticulum Ca2+ release through ryanodine and inositol 1,4,5-triphosphate receptors in the neurotoxic effects induced by the amyloid-beta peptide.内质网通过兰尼碱受体和肌醇1,4,5-三磷酸受体释放Ca2+参与β-淀粉样肽诱导的神经毒性作用。
J Neurosci Res. 2004 Jun 15;76(6):872-80. doi: 10.1002/jnr.20135.
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Internalized Amyloid-β (1-42) Peptide Inhibits the Store-Operated Calcium Entry in HT-22 Cells.内源性淀粉样β（1-42）肽抑制 HT-22 细胞的钙库操纵性钙内流。
Int J Mol Sci. 2022 Oct 21;23(20):12678. doi: 10.3390/ijms232012678.
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The Relevance of Amyloid β-Calmodulin Complexation in Neurons and Brain Degeneration in Alzheimer's Disease.淀粉样β-钙调蛋白复合物在神经元中的相关性及阿尔茨海默病中的脑退行性变。
Int J Mol Sci. 2021 May 7;22(9):4976. doi: 10.3390/ijms22094976.
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Alterations of the Endoplasmic Reticulum (ER) Calcium Signaling Molecular Components in Alzheimer's Disease.阿尔茨海默病中内质网钙信号分子成分的改变。
Cells. 2020 Dec 1;9(12):2577. doi: 10.3390/cells9122577.
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Reversal of Calcium Dysregulation as Potential Approach for Treating Alzheimer's Disease.钙稳态失衡的逆转：治疗阿尔茨海默病的潜在方法。
Curr Alzheimer Res. 2020;17(4):344-354. doi: 10.2174/1567205017666200528162046.
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Neuronal Ryanodine Receptors in Development and Aging.发育与衰老过程中的神经元兰尼碱受体。
Mol Neurobiol. 2018 Feb;55(2):1183-1192. doi: 10.1007/s12035-016-0375-4. Epub 2017 Jan 19.
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Ryanodine receptors: physiological function and deregulation in Alzheimer disease.兰尼碱受体：阿尔茨海默病中的生理功能与失调。
Mol Neurodegener. 2014 Jun 5;9:21. doi: 10.1186/1750-1326-9-21.
本文引用的文献
1
Canonical Wnt signaling protects hippocampal neurons from Aβ oligomers: role of non-canonical Wnt-5a/Ca(2+) in mitochondrial dynamics.经典 Wnt 信号通路可保护海马神经元免受 Aβ寡聚体的侵害：非经典 Wnt-5a/Ca(2+)在线粒体动力学中的作用。
Front Cell Neurosci. 2013 Jun 25;7:97. doi: 10.3389/fncel.2013.00097. eCollection 2013.
2
Deregulation of excitatory neurotransmission underlying synapse failure in Alzheimer's disease.阿尔茨海默病中突触衰竭的兴奋性神经递质调节失控。
J Neurochem. 2013 Jul;126(2):191-202. doi: 10.1111/jnc.12304. Epub 2013 May 28.
3
The CAMKK2-AMPK kinase pathway mediates the synaptotoxic effects of Aβ oligomers through Tau phosphorylation.CAMKK2-AMPK 激酶通路通过 Tau 磷酸化介导 Aβ 寡聚物的突触毒性作用。
Neuron. 2013 Apr 10;78(1):94-108. doi: 10.1016/j.neuron.2013.02.003.
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Mitochondrial dynamics in neurodegeneration.线粒体在神经变性中的动态变化。
Trends Cell Biol. 2013 Feb;23(2):64-71. doi: 10.1016/j.tcb.2012.10.006. Epub 2012 Nov 16.
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A role for calcineurin in Alzheimer's disease.钙调神经磷酸酶在阿尔茨海默病中的作用。
Curr Neuropharmacol. 2011 Dec;9(4):685-92. doi: 10.2174/157015911798376316.
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Impaired mitochondrial function in psychiatric disorders.精神疾病中线粒体功能障碍。
Nat Rev Neurosci. 2012 Apr 18;13(5):293-307. doi: 10.1038/nrn3229.
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The antioxidant N-acetylcysteine prevents the mitochondrial fragmentation induced by soluble amyloid-β peptide oligomers.抗氧化剂 N-乙酰半胱氨酸可预防可溶性淀粉样β肽寡聚物诱导的线粒体碎片化。
Neurodegener Dis. 2012;10(1-4):34-7. doi: 10.1159/000334901. Epub 2012 Jan 24.
8
The Aβ oligomer hypothesis for synapse failure and memory loss in Alzheimer's disease.阿尔茨海默病中突触衰竭和记忆丧失的 Aβ 寡聚物假说。
Neurobiol Learn Mem. 2011 Nov;96(4):529-43. doi: 10.1016/j.nlm.2011.08.003. Epub 2011 Sep 6.
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Regulation of NMDA-receptor synaptic transmission by Wnt signaling.Wnt 信号对 NMDA 受体突触传递的调节。
J Neurosci. 2011 Jun 29;31(26):9466-71. doi: 10.1523/JNEUROSCI.6311-10.2011.
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Aberrant mitochondrial fission in neurons induced by protein kinase C{delta} under oxidative stress conditions in vivo.体内氧化应激条件下蛋白激酶 C{delta}诱导神经元中线粒体的异常分裂。
Mol Biol Cell. 2011 Jan 15;22(2):256-65. doi: 10.1091/mbc.E10-06-0551. Epub 2010 Nov 30.
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