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The origin of B cell recurrent chromosomal translocations: proximity versus DNA damage.
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Response to Casellas et al.
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Close proximity to Igh is a contributing factor to AID-mediated translocations.
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Role of genomic instability and p53 in AID-induced c-myc-Igh translocations.
Nature. 2006 Mar 2;440(7080):105-9. doi: 10.1038/nature04495. Epub 2006 Jan 8.
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DNA damage defines sites of recurrent chromosomal translocations in B lymphocytes.
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Restricting activation-induced cytidine deaminase tumorigenic activity in B lymphocytes.
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AID is required for the chromosomal breaks in c-myc that lead to c-myc/IgH translocations.
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Mechanisms promoting translocations in editing and switching peripheral B cells.
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DSB structure impacts DNA recombination leading to class switching and chromosomal translocations in human B cells.
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Transcription factor binding at Ig enhancers is linked to somatic hypermutation targeting.
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2
Mechanisms of human lymphoid chromosomal translocations.
Nat Rev Cancer. 2016 May 25;16(6):387-98. doi: 10.1038/nrc.2016.40.

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1
RPA accumulation during class switch recombination represents 5'-3' DNA-end resection during the S-G2/M phase of the cell cycle.
Cell Rep. 2013 Jan 31;3(1):138-47. doi: 10.1016/j.celrep.2012.12.006. Epub 2013 Jan 3.
2
Close proximity to Igh is a contributing factor to AID-mediated translocations.
Mol Cell. 2012 Sep 28;47(6):873-85. doi: 10.1016/j.molcel.2012.06.036. Epub 2012 Aug 2.
4
DNA damage defines sites of recurrent chromosomal translocations in B lymphocytes.
Nature. 2012 Feb 7;484(7392):69-74. doi: 10.1038/nature10909.
8
Comprehensive mapping of long-range interactions reveals folding principles of the human genome.
Science. 2009 Oct 9;326(5950):289-93. doi: 10.1126/science.1181369.
9
An evaluation of 3C-based methods to capture DNA interactions.
Nat Methods. 2007 Nov;4(11):895-901. doi: 10.1038/nmeth1114.

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