Brown and Goldstein: the cholesterol chronicles.
作者信息
Nair Prashant
出版信息
Proc Natl Acad Sci U S A. 2013 Sep 10;110(37):14829-32. doi: 10.1073/pnas.1315180110. Epub 2013 Aug 26.
Abstract
摘要
相似文献
1
Brown and Goldstein: the cholesterol chronicles.布朗和戈尔茨坦：胆固醇编年史
Proc Natl Acad Sci U S A. 2013 Sep 10;110(37):14829-32. doi: 10.1073/pnas.1315180110. Epub 2013 Aug 26.
2
Discovery of the cellular and molecular basis of cholesterol control.胆固醇代谢调控的细胞与分子基础研究进展
Proc Natl Acad Sci U S A. 2013 Sep 10;110(37):14833-6. doi: 10.1073/pnas.1312967110. Epub 2013 Aug 23.
3
Presentation of the Kober Medal to Joseph L. Goldstein and Michael S. Brown.向约瑟夫·L·戈尔茨坦和迈克尔·S·布朗颁发科伯奖章。
J Clin Invest. 2002 Dec;110(12):S5-9. doi: 10.1172/jci120039.
4
The LDL receptor.低密度脂蛋白受体
Arterioscler Thromb Vasc Biol. 2009 Apr;29(4):431-8. doi: 10.1161/ATVBAHA.108.179564.
5
Lowering plasma cholesterol by raising ldl receptors. 1981.
Atheroscler Suppl. 2004 Oct;5(3):57-9. doi: 10.1016/j.atherosclerosissup.2004.08.024.
6
Nutrition classics. Proceedings of the National Academy of Sciences of the United States of America, Volume 71, 1974: Familial hypercholesterolemia: defective binding of lipoproteins to cultured fibroblasts associated with impaired regulation of 3-hydroxy-3-methylglutaryl coenzyme A reductase activity. By Michael S. Brown and Joseph L. Goldstein.
Nutr Rev. 1985 Sep;43(9):274-8. doi: 10.1111/j.1753-4887.1985.tb02448.x.
7
Mevinolin and colestipol stimulate receptor-mediated clearance of low density lipoprotein from plasma in familial hypercholeserolemia heterozygotes. 1983.美维诺林和考来替泊可刺激家族性高胆固醇血症杂合子血浆中低密度脂蛋白的受体介导清除。1983年。
Atheroscler Suppl. 2004 Oct;5(3):61-5. doi: 10.1016/j.atherosclerosissup.2004.08.025.
8
Editor's Preamble to the Profile of Joseph Goldstein and Michael Brown.约瑟夫·戈德斯坦和迈克尔·布朗简介的编辑前言。
Circ Res. 2015 Oct 9;117(9):e68. doi: 10.1161/CIRCRESAHA.115.307566.
9
Obituary of Dr N.B. Myant.N.B. 迈恩特博士讣告。
Atherosclerosis. 2015 Jun;240(2):437-8. doi: 10.1016/j.atherosclerosis.2015.03.035.
10
Molecular medicine. The cholesterol quartet.分子医学。胆固醇四重奏。
Science. 2001 May 18;292(5520):1310-2. doi: 10.1126/science.1061815.
引用本文的文献
1
Statins in the Cause and Prevention of Cancer: Confounding by Indication and Mediation by Rhabdomyolysis and Phosphate Toxicity.他汀类药物在癌症病因及预防中的作用：指征性混杂以及横纹肌溶解和磷酸盐毒性介导作用
J Cardiovasc Dev Dis. 2024 Sep 23;11(9):296. doi: 10.3390/jcdd11090296.
2
Single case designs for early phase behavioral translational research in health psychology.健康心理学早期行为转化研究的单病例设计
Health Psychol. 2021 Dec;40(12):858-874. doi: 10.1037/hea0001055. Epub 2021 Aug 9.
3
An Allosteric Binding Site on Sortilin Regulates the Trafficking of VLDL, PCSK9, and LDLR in Hepatocytes.Sortilin 的别构结合位点调节肝细胞中 VLDL、PCSK9 和 LDLR 的转运。
Biochemistry. 2020 Nov 17;59(45):4321-4335. doi: 10.1021/acs.biochem.0c00741. Epub 2020 Nov 5.
4
NHLBI Strategic Visioning: setting an agenda together for the NHLBI of 2025.美国国立心肺血液研究所战略展望：共同为2025年的美国国立心肺血液研究所制定议程。
Blood. 2015 Apr 23;125(17):2733-5. doi: 10.1182/blood-2015-02-626689.
5
National Heart, Lung, and Blood Institute Strategic Visioning: setting an agenda together for the NHLBI of 2025.美国国立心肺血液研究所战略展望：共同为2025年的国立心肺血液研究所制定议程。
Am J Public Health. 2015 May;105(5):e25-8. doi: 10.2105/AJPH.2015.302605. Epub 2015 Feb 27.
6
NHLBI strategic visioning: setting an agenda together for the NHLBI of 2025.美国国立心肺血液研究所战略展望：共同为2025年的美国国立心肺血液研究所制定议程。
Am J Respir Crit Care Med. 2015 Mar 1;191(5):489-91. doi: 10.1164/rccm.201501-0194ED.
本文引用的文献
1
Sequence variations in PCSK9, low LDL, and protection against coronary heart disease.前蛋白转化酶枯草溶菌素9（PCSK9）的序列变异、低密度脂蛋白水平降低与冠心病预防
N Engl J Med. 2006 Mar 23;354(12):1264-72. doi: 10.1056/NEJMoa054013.
2
ER stress induces cleavage of membrane-bound ATF6 by the same proteases that process SREBPs.内质网应激通过处理固醇调节元件结合蛋白（SREBPs）的相同蛋白酶诱导膜结合型激活转录因子6（ATF6）的裂解。
Mol Cell. 2000 Dec;6(6):1355-64. doi: 10.1016/s1097-2765(00)00133-7.
3
The presenilins in Alzheimer's disease--proteolysis holds the key.阿尔茨海默病中的早老素——蛋白水解是关键。
Science. 1999 Oct 29;286(5441):916-9. doi: 10.1126/science.286.5441.916.
4
A presenilin-1-dependent gamma-secretase-like protease mediates release of Notch intracellular domain.一种早老素-1依赖性γ-分泌酶样蛋白酶介导Notch细胞内结构域的释放。
Nature. 1999 Apr 8;398(6727):518-22. doi: 10.1038/19083.
5
The SREBP pathway: regulation of cholesterol metabolism by proteolysis of a membrane-bound transcription factor.固醇调节元件结合蛋白（SREBP）途径：通过膜结合转录因子的蛋白水解作用调节胆固醇代谢
Cell. 1997 May 2;89(3):331-40. doi: 10.1016/s0092-8674(00)80213-5.
6
Familial hypercholesterolemia: defective binding of lipoproteins to cultured fibroblasts associated with impaired regulation of 3-hydroxy-3-methylglutaryl coenzyme A reductase activity.家族性高胆固醇血症：脂蛋白与培养的成纤维细胞结合缺陷，与3-羟基-3-甲基戊二酰辅酶A还原酶活性调节受损有关。
Proc Natl Acad Sci U S A. 1974 Mar;71(3):788-92. doi: 10.1073/pnas.71.3.788.
7
A mutation that impairs the ability of lipoprotein receptors to localise in coated pits on the cell surface of human fibroblasts.
Nature. 1977;270(5639):695-9. doi: 10.1038/270695a0.
8
Analysis of a mutant strain of human fibroblasts with a defect in the internalization of receptor-bound low density lipoprotein.对一种人类成纤维细胞突变株的分析，该突变株在受体结合型低密度脂蛋白的内化过程中存在缺陷。
Cell. 1976 Dec;9(4 PT 2):663-74. doi: 10.1016/0092-8674(76)90130-6.
Zotero 插件