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Challenges and opportunities of sensory plasticity after SCI.

作者信息

Petruska Jeffrey C, Hubscher Charles H, Rabchevsky Alexander G

机构信息

Department of Anatomical Sciences and Neurobiology, Kentucky Spinal Cord Injury Research Center, University of Louisville Louisville, KY, USA.

出版信息

Front Physiol. 2013 Aug 27;4:231. doi: 10.3389/fphys.2013.00231. eCollection 2013.

DOI:10.3389/fphys.2013.00231
PMID:23986722
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3753431/
Abstract
摘要

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Delayed Exercise Is Ineffective at Reversing Aberrant Nociceptive Afferent Plasticity or Neuropathic Pain After Spinal Cord Injury in Rats.延迟运动对逆转大鼠脊髓损伤后异常的伤害性传入可塑性或神经性疼痛无效。
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本文引用的文献

1
The Transcriptional Response of Neurotrophins and Their Tyrosine Kinase Receptors in Lumbar Sensorimotor Circuits to Spinal Cord Contusion is Affected by Injury Severity and Survival Time.神经生长因子及其酪氨酸激酶受体在脊髓挫伤后腰感觉运动回路中的转录反应受损伤严重程度和存活时间的影响。
Front Physiol. 2013 Jan 9;3:478. doi: 10.3389/fphys.2012.00478. eCollection 2012.
2
Maladaptive spinal plasticity opposes spinal learning and recovery in spinal cord injury.适应性不良的脊髓可塑性阻碍了脊髓损伤中的脊髓学习和恢复。
Front Physiol. 2012 Oct 10;3:399. doi: 10.3389/fphys.2012.00399. eCollection 2012.
3
Central nociceptive sensitization vs. spinal cord training: opposing forms of plasticity that dictate function after complete spinal cord injury.中枢性伤害感受过敏与脊髓训练：完全性脊髓损伤后决定功能的两种相反形式的可塑性。
Front Physiol. 2012 Oct 4;3:396. doi: 10.3389/fphys.2012.00396. eCollection 2012.
4
Plasticity in ascending long propriospinal and descending supraspinal pathways in chronic cervical spinal cord injured rats.慢性颈脊髓损伤大鼠中长距离脊髓固有束上行和脊髓上运动神经元下行通路的可塑性
Front Physiol. 2012 Aug 17;3:330. doi: 10.3389/fphys.2012.00330. eCollection 2012.
5
Effects of gabapentin on muscle spasticity and both induced as well as spontaneous autonomic dysreflexia after complete spinal cord injury.加巴喷丁对完全性脊髓损伤后肌肉痉挛以及诱发性和自发性自主神经反射异常的影响。
Front Physiol. 2012 Aug 15;3:329. doi: 10.3389/fphys.2012.00329. eCollection 2012.
6
Nociceptors as chronic drivers of pain and hyperreflexia after spinal cord injury: an adaptive-maladaptive hyperfunctional state hypothesis.伤害感受器作为脊髓损伤后疼痛和反射亢进的慢性驱动因素：一种适应性 - 适应不良的高功能状态假说
Front Physiol. 2012 Aug 2;3:309. doi: 10.3389/fphys.2012.00309. eCollection 2012.
7
Upper gastrointestinal dysmotility after spinal cord injury: is diminished vagal sensory processing one culprit?脊髓损伤后上消化道动力障碍：迷走神经感觉处理功能减退是罪魁祸首之一吗？
Front Physiol. 2012 Jul 17;3:277. doi: 10.3389/fphys.2012.00277. eCollection 2012.
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Impact of behavioral control on the processing of nociceptive stimulation.行为控制对伤害性刺激处理的影响。
Front Physiol. 2012 Aug 10;3:262. doi: 10.3389/fphys.2012.00262. eCollection 2012.
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Altered patterns of reflex excitability, balance, and locomotion following spinal cord injury and locomotor training.脊髓损伤及运动训练后反射兴奋性、平衡和运动模式的改变。
Front Physiol. 2012 Jul 18;3:258. doi: 10.3389/fphys.2012.00258. eCollection 2012.
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Plasticity of TRPV1-Expressing Sensory Neurons Mediating Autonomic Dysreflexia Following Spinal Cord Injury.脊髓损伤后介导自主神经反射异常的表达TRPV1的感觉神经元的可塑性
Front Physiol. 2012 Jul 9;3:257. doi: 10.3389/fphys.2012.00257. eCollection 2012.