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[猫失血性休克时脂肪细胞质膜的磷脂代谢]

[The phospholipid metabolism of adipocyte plasma membranes in hemorrhagic shock in cats].

作者信息

Leskova G F, Su L B, Udovichenko V I

出版信息

Patol Fiziol Eksp Ter. 1990 May-Jun(3):34-5.

PMID:2399036
Abstract

The mechanisms of functional disorders in adipocytes were studied on models of hemorrhagic shock. Injection of heparin as an anticoagulant caused regional differences in animals in phosphatidylethanolamine metabolism in the plasma membranes of adipocytes of the subcutaneous fat and those of the abdominal fat (omentum). The disorders of phospholipid metabolism in the plasma membranes of adipocytes of the subcutaneous and abdominal fat in hemorrhagic shock were unidirectional and associated with intensified breakdown of phosphatidylinositol.

摘要

在失血性休克模型上研究了脂肪细胞功能紊乱的机制。注射肝素作为抗凝剂导致动物皮下脂肪和腹部脂肪(网膜)脂肪细胞质膜中磷脂酰乙醇胺代谢出现区域差异。失血性休克时皮下和腹部脂肪脂肪细胞质膜中磷脂代谢紊乱是单向的,且与磷脂酰肌醇的分解加剧有关。

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