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[眼屈光间质透明度障碍的发病机制。VI. 晶状体铜中毒的透射电子显微镜观察]

[Pathogenesis of disorders of transparency of the optic media of the eye. VI. Transmission electron microscopy in chalcosis of the lens].

作者信息

Moster M F, Synek S, Pác L

机构信息

Oftalmologická biofyzikální laborator, ocní klinika LF UJEP, Brno.

出版信息

Cesk Oftalmol. 1990 Jul;46(4):276-84.

PMID:2401024
Abstract

By means of transmission electron microscopy in the chalcotic lens ultrastructural changes were probed which can be divided into specific and non-specific ones. Specific findings include proliferation of vacuoles in the cytoplasm, development of oedema of lenticular fibres and dilatation of intercellular spaces. For specific findings intercellular chalcosomes in the shape of copper crystals are typical. Copper crystals are dispersed also in the cytoplasm in formations lined by the membrane. Non-specific changes in the chalcotic lens can be classified as degenerative changes in the lenticular capsule, which is divided into segments.

摘要

通过透射电子显微镜对铜中毒晶状体的超微结构变化进行了探究,这些变化可分为特异性和非特异性变化。特异性表现包括细胞质中液泡的增殖、晶状体纤维水肿的形成以及细胞间隙的扩张。典型的特异性表现是呈铜晶体形状的细胞间铜质小体。铜晶体也以膜包被的形式分散在细胞质中。铜中毒晶状体的非特异性变化可归类为晶状体囊膜的退行性变化,晶状体囊膜会分成节段。

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