Magrotti E, Borutti G, Mariani G, Donati E, Faggi L
Division of Neurology, U.S.S.L. 79, Voghera, Italy.
Funct Neurol. 1990 Jan-Mar;5(1):65-71.
Twenty patients suffering from ataxic hemiparesis caused by lacunar infarcts were studied using computerized tomography. Supratentorial lesions were found in nineteen patients (95%) while subtentorial lesion was found in only one patient (5%). In all cases the lesions were localized along the corticopontine pathways, making the hypothesis of injury to this pathway the most valid explanation of ataxia.