Adachi M, Hosoya T, Yamaguchi K
Department of Radiology, Nagai Municipal Hospital, Japan.
Nihon Igaku Hoshasen Gakkai Zasshi. 1996 Apr;56(5):279-82.
Although lacunar syndrome is relatively commonly encountered in patients with a lacunar infarct in the deep cerebral white matter, the responsible lesions are often unidentified. By examining eight patients with lacunar syndrome who had small infarcts in the deep cerebral white matter, an attempt was made to determine the position and distribution of the sensory pathway on brain CT at the level of the splenium of the corpus callosum. When the distance from the frontal crista to the superior saggital sinus sulcus was defined as 1.0, the position of the corticospinal tract was located in the portion from 0.45 to 0.49 and that of the sensory pathway in the portion from 0.49 to 0.53. These data indicated that the position of the sensory pathway was just behind the corticospinal tract and the distribution was very narrow at this level. The results of this assessment revealed that lacunar infarcts in the deep cerebral white matter were liable to cause sensorimoter syndrome more than pure sensory stroke.
虽然腔隙综合征在大脑深部白质发生腔隙性梗死的患者中相对常见,但责任病灶往往难以确定。通过对8例大脑深部白质有小梗死灶的腔隙综合征患者进行检查,试图确定胼胝体压部层面脑CT上感觉通路的位置和分布。当将额嵴至矢状窦上沟的距离定义为1.0时,皮质脊髓束的位置位于0.45至0.49的部分,感觉通路的位置位于0.49至0.53的部分。这些数据表明,感觉通路的位置恰好在皮质脊髓束的后方,且在此层面分布非常狭窄。该评估结果显示,大脑深部白质的腔隙性梗死比单纯感觉性卒中更容易导致感觉运动综合征。