From the Department of Anesthesiology and Pain Medicine, Seoul National University Hospital; Department of Anesthesiology and Pain Medicine, Samsung Medical Center; and Department of radiology, Seoul National University Hospital, Seoul, Korea.
Anesth Analg. 2013 Oct;117(4):1017-1021. doi: 10.1213/ANE.0b013e3182a1ee53. Epub 2013 Sep 10.
For unilateral spinal block, local anesthetics should affect the spinal nerves of 1 side. With full flexion of the spine, the sunken cauda equina becomes tightened and is suspended in the middle of the subarachnoid space. We performed this study to assess whether spinal flexion facilitates unilateral spinal anesthesia.
Hyperbaric bupivacaine (8 mg) was administered at the L3-4 interspace through a 25-gauge Quincke needle at a rate of 0.02 mL/s. Patients were randomly allocated to group F (with full spinal flexion) or group N (the hips and back straightened). After maintaining the lateral position for 15 minutes with or without spinal flexion, patients were gently returned to the supine position. Spinal blockade was assessed by loss of pinprick sensation and the modified Bromage motor scale.
While the lateral position was maintained, sensory block was noted on the nondependent side in 14 of 16 patients in group N (87.5%) but only in 1 of 16 patients in group F (6.3%) (P < 0.001). The median level of sensory block in group N was L5 on the nondependent side just before turning to the supine position. When patients were returned to the supine position, sensory blockade on the nondependent side was noted in all group N patients (100%) and 15 group F patients (93.7%). The sensory level on the nondependent side between group N and group F were similar after turning supine.
Strict unilateral sensory block was not achieved even after lateral decubitus positioning with spinal flexion, when 8 mg hyperbaric bupivacaine was administered manually at a conventionally slow rate through a beveled spinal needle. However, maintaining flexion of the spinal column during lateral decubitus positioning altered the initial onset of sensory block with respect to laterality.
对于单侧脊髓阻滞,局部麻醉剂应影响 1 侧的脊髓神经。脊柱完全弯曲时,凹陷的马尾神经收紧并悬停在蛛网膜下腔中间。我们进行这项研究是为了评估脊柱弯曲是否有助于单侧脊髓麻醉。
在 L3-4 间隙通过 25 号 Quincke 针以 0.02 毫升/秒的速度给予 8 毫克重比重布比卡因。患者随机分配到 F 组(完全脊柱弯曲)或 N 组(臀部和背部伸直)。在保持侧卧位 15 分钟,有或没有脊柱弯曲后,患者被轻轻返回到仰卧位。通过感觉丧失和改良 Bromage 运动量表评估脊髓阻滞。
在保持侧卧位时,N 组 16 例患者中有 14 例(87.5%)在非依赖侧出现感觉阻滞,但 F 组只有 1 例(6.3%)(P<0.001)。N 组非依赖侧感觉阻滞的中位数水平在转为仰卧位前为 L5。当患者返回到仰卧位时,N 组所有患者(100%)和 15 例 F 组患者(93.7%)均出现非依赖侧感觉阻滞。转为仰卧位后,N 组和 F 组非依赖侧的感觉阻滞水平相似。
当手动给予 8 毫克重比重布比卡因通过斜面针以常规缓慢速度给药时,即使在脊柱弯曲的侧卧位下,也未达到严格的单侧感觉阻滞。然而,在侧卧位期间保持脊柱弯曲改变了感觉阻滞的初始出现与偏侧性的关系。