Wenger Markus, Markwalder Thomas-Marc
Department of Neurosurgery, Klinik Beau-Site and Salemspital, Schänzlihalde 11, CH-3000 Bern 25, Switzerland.
Neurosurgeon FMH, Private Practice Spine Surgery, CH-3123 Bern-Belp, Switzerland.
J Clin Neurosci. 2014 May;21(5):741-4. doi: 10.1016/j.jocn.2013.07.016. Epub 2013 Sep 11.
We report on 69 retrospectively reviewed patients who received 73 Bryan (Medtronic Sofamor Danek, Memphis, TN, USA) total disc arthroplasties for recent soft cervical disc herniations over a 9.3year period. Three patients returned with radiculopathy due to the redevelopment of uncoforaminal stenosis at the Bryan segment and later underwent posterior decompression of the uncoforaminal area without modification to the prosthesis. They recovered from the radiculopathy after decompression; however, one patient later required adjacent segment fusion to recover from concomitant cervicalgia. After posterior decompression, all prostheses continued to function normally. In one patient, however, bony bridging of the prosthesis is imminent, despite being currently asymptomatic. We normally exclude patients with uncoforaminal stenosis from Bryan arthroplasty. Analysis of three of these patients (4.3% of patients, 4.1% of prostheses) revealed that they received a prosthesis despite slight uncoforaminal stenosis (slight stenosis was known prior to surgery in one instance, two others were only discovered intra-operatively). Our observation raises the suspicion that slight uncoforaminal stenosis could also recur in physiologically working arthroplasty segments, and that in some instances this spur formation may progress into prosthesis bridging. However, more research is required to confirm the significance of uncoforaminal stenosis discovered pre- or intra-operatively in arthroplasty patients. Posterior minimally invasive decompression using the Frykholm-Scoville keyhole approach successfully treats uncoforaminal stenosis without revising the prosthesis.
我们报告了69例接受回顾性研究的患者,这些患者在9.3年的时间里因近期软性颈椎间盘突出症接受了73例Bryan(美敦力索法玛丹尼克公司,美国田纳西州孟菲斯)全椎间盘置换术。3例患者因Bryan节段钩椎孔狭窄复发而出现神经根病,随后接受了钩椎孔区域的后路减压,假体未作修改。减压后他们从神经根病中康复;然而,1例患者后来需要进行相邻节段融合以缓解伴随的颈部疼痛。后路减压后,所有假体仍继续正常发挥功能。然而,有1例患者尽管目前无症状,但假体的骨桥形成即将发生。我们通常将钩椎孔狭窄患者排除在Bryan置换术之外。对其中3例患者(占患者总数的4.3%,占假体总数的4.1%)的分析显示,尽管存在轻微的钩椎孔狭窄(其中1例在手术前已知有轻微狭窄,另外2例仅在术中发现),他们仍接受了假体植入。我们的观察引发了一种怀疑,即轻微的钩椎孔狭窄在生理功能正常的置换节段中也可能复发,并且在某些情况下,这种骨刺形成可能会发展为假体桥接。然而,需要更多的研究来证实术前或术中发现的钩椎孔狭窄在置换术患者中的意义。采用Frykholm-Scoville锁孔入路进行后路微创减压可成功治疗钩椎孔狭窄,而无需翻修假体。