再灌注早期短暂酸中毒通过 PI3k-Akt-eNOS 信号通路减轻心肌缺血再灌注损伤。
Transient acidosis during early reperfusion attenuates myocardium ischemia reperfusion injury via PI3k-Akt-eNOS signaling pathway.
机构信息
Department of Anesthesiology, Chongqing Zhoushan Hospital, Chongqing 404100, China ; Department of Anesthesiology, The First Affiliated Hospital of Chongqing Medical University, Chongqing 404100, China.
出版信息
Oxid Med Cell Longev. 2013;2013:126083. doi: 10.1155/2013/126083. Epub 2013 Nov 7.
Abstract
In this paper, we concluded that transient acidosis reperfusion conferred cardioprotection against myocardial ischemia reperfusion injury in isolated rat hearts through activating PI3K-Akt-eNOS pathway.
摘要
在本论文中，我们的结论为：短暂性酸中毒再灌注通过激活 PI3K-Akt-eNOS 通路，为离体鼠心的心肌缺血再灌注损伤提供了心脏保护作用。
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