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膀胱过度活动症综合征以及前列腺素和磷酸二酯酶的潜在作用:引言

Overactive bladder syndrome and the potential role of prostaglandins and phosphodiesterases: an introduction.

作者信息

Rahnama'i Mohammad Sajjad, Van Koeveringe Gommert A, Van Kerrebroeck Philip E

机构信息

Maastricht University Medical Centre (MUMC+), Maastricht, The Netherlands.

出版信息

Nephrourol Mon. 2013 Sep;5(4):934-45. doi: 10.5812/numonthly.14087. Epub 2013 Sep 10.

Abstract

In this paper, a general introduction is given, presenting the overactive bladder syndrome (OAB) and its impact on the quality of life and economical burden in patients affected. Moreover, the anatomy, physiology and histology of the lower urinary tract are discussed, followed by a brief overview on the possible role of prostaglandin (PG) and phosphodiesterase type 5 (PDE5) in the urinary bladder. The current literature on the role and distribution of PGE2 and its receptors in the urinary bladder is discussed. In both animal models and in human studies, high levels of signaling molecules such as PG and cGMP have been implicated, in decreased functional bladder capacity and micturition volume, as well as in increased voiding contraction amplitude. As a consequence, inhibition of prostanoid production, the use of prostanoid receptor antagonists, or PDE inhibitors might be a rational way to treat patients with detrusor overactivity. Similarly, prostanoid receptor agonists, or agents that stimulate their production, might have a function in treating bladder underactivity.

摘要

本文进行了全面介绍,阐述了膀胱过度活动症(OAB)及其对受影响患者生活质量和经济负担的影响。此外,还讨论了下尿路的解剖学、生理学和组织学,随后简要概述了前列腺素(PG)和5型磷酸二酯酶(PDE5)在膀胱中的可能作用。讨论了关于PGE2及其受体在膀胱中的作用和分布的现有文献。在动物模型和人体研究中,已表明高水平的信号分子如PG和cGMP与膀胱功能容量和排尿量减少以及排尿收缩幅度增加有关。因此,抑制前列腺素生成、使用前列腺素受体拮抗剂或PDE抑制剂可能是治疗逼尿肌过度活动患者的合理方法。同样,前列腺素受体激动剂或刺激其生成的药物可能在治疗膀胱活动不足方面发挥作用。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/51a7/3842572/b6324698be73/num-05-934-i001.jpg

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