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Notch3 marks clonogenic mammary luminal progenitor cells in vivo.
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Aurora A kinase regulates mammary epithelial cell fate by determining mitotic spindle orientation in a Notch-dependent manner.
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Chromatin effector Pygo2 mediates Wnt-notch crosstalk to suppress luminal/alveolar potential of mammary stem and basal cells.
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Notch2 genetic fate mapping reveals two previously unrecognized mammary epithelial lineages.
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Global changes in the mammary epigenome are induced by hormonal cues and coordinated by Ezh2.
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Developmental stage and time dictate the fate of Wnt/β-catenin-responsive stem cells in the mammary gland.
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Lunatic fringe deficiency cooperates with the Met/Caveolin gene amplicon to induce basal-like breast cancer.
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Distinct stem cells contribute to mammary gland development and maintenance.
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Wnt proteins are self-renewal factors for mammary stem cells and promote their long-term expansion in culture.
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