Groves Elliott M, Kim Jin Kyung
Division of Cardiology, Department of Medicine, University of California at Irvine, 333 City Blvd West, Suite 400, Orange, CA 92868, USA ; Edwards Lifesciences Center for Advanced Cardiovascular Technology, Department of Bioengineering, University of California at Irvine, 5200 Engineering Hall, Irvine, CA 92697, USA.
Division of Cardiology, Department of Medicine, University of California at Irvine, 333 City Blvd West, Suite 400, Orange, CA 92868, USA ; UC Irvine Medical Center, 101 The City Drive, Orange, CA 92868, USA.
Case Rep Med. 2013;2013:106757. doi: 10.1155/2013/106757. Epub 2013 Nov 28.
Importance. Constrictive pericarditis is a rare clinical entity that frequently necessitates surgical intervention. Here we present a case of biventricular failure due to stress cardiomyopathy after pericardiectomy. This is an extremely rare complication that is not well described and does not have a definitive mechanism. Observations. A 40-year-old Ecuadorian woman who was found to have constrictive pericarditis due to Mycobacterium tuberculosis infection was referred to our institution. The presence of constrictive pericarditis was confirmed by echocardiography, computed tomography, magnetic resonance imaging, and cardiac catheterization. Following pericardiectomy, the patient developed biventricular failure consistent with stress cardiomyopathy (Takotsubo cardiomyopathy), based on the echocardiographic assessment of the ventricles, which demonstrated an akinetic apex and hyperactive base in both ventricles, the absence of significant epicardial coronary atherosclerosis, and prompt normalization of the cardiac function after intensive medical therapy. Conclusions and Relevance. Biventricular failure in the form of stress cardiomyopathy after pericardiectomy in the manner presented here has not been previously described in the literature. While postulations as to the cause of single ventricle dysfunction have been described, the exact mechanism is unclear and current theories do not explain the clinical features in this case of stress cardiomyopathy after pericardiectomy.
重要性。缩窄性心包炎是一种罕见的临床病症,常常需要手术干预。在此,我们报告一例心包切除术后因应激性心肌病导致双心室衰竭的病例。这是一种极为罕见的并发症,目前描述甚少,且尚无确切机制。病例观察。一名40岁的厄瓜多尔女性因结核分枝杆菌感染被诊断为缩窄性心包炎,转诊至我院。通过超声心动图、计算机断层扫描、磁共振成像和心导管检查确诊为缩窄性心包炎。心包切除术后,根据心室的超声心动图评估,患者出现了与应激性心肌病(Takotsubo心肌病)相符的双心室衰竭,表现为双心室心尖运动减弱、心底运动增强,无明显的心外膜冠状动脉粥样硬化,且在强化药物治疗后心功能迅速恢复正常。结论及相关性。本文所呈现的心包切除术后以应激性心肌病形式出现的双心室衰竭在以往文献中未曾描述。虽然已有关于单心室功能障碍病因的推测,但确切机制尚不清楚,目前的理论也无法解释该例心包切除术后应激性心肌病的临床特征。