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1
ALS-linked mutations enlarge TDP-43-enriched neuronal RNA granules in the dendritic arbor.
J Neurosci. 2014 Mar 19;34(12):4167-74. doi: 10.1523/JNEUROSCI.2350-13.2014.
2
Amyotrophic lateral sclerosis-linked mutations increase the viscosity of liquid-like TDP-43 RNP granules in neurons.
Proc Natl Acad Sci U S A. 2017 Mar 21;114(12):E2466-E2475. doi: 10.1073/pnas.1614462114. Epub 2017 Mar 6.
3
TDP-43 aggregation in neurodegeneration: are stress granules the key?
Brain Res. 2012 Jun 26;1462:16-25. doi: 10.1016/j.brainres.2012.02.032. Epub 2012 Feb 22.
4
USP10 Inhibits Aberrant Cytoplasmic Aggregation of TDP-43 by Promoting Stress Granule Clearance.
Mol Cell Biol. 2022 Mar 17;42(3):e0039321. doi: 10.1128/MCB.00393-21. Epub 2022 Jan 10.
6
Endogenous TDP-43, but not FUS, contributes to stress granule assembly via G3BP.
Mol Neurodegener. 2012 Oct 24;7:54. doi: 10.1186/1750-1326-7-54.
7
TDP-43 is directed to stress granules by sorbitol, a novel physiological osmotic and oxidative stressor.
Mol Cell Biol. 2011 Mar;31(5):1098-108. doi: 10.1128/MCB.01279-10. Epub 2010 Dec 20.
8
Altered mRNP granule dynamics in FTLD pathogenesis.
J Neurochem. 2016 Aug;138 Suppl 1:112-33. doi: 10.1111/jnc.13601. Epub 2016 Jun 15.
10
TDP-43, the signature protein of FTLD-U, is a neuronal activity-responsive factor.
J Neurochem. 2008 May;105(3):797-806. doi: 10.1111/j.1471-4159.2007.05190.x. Epub 2007 Dec 15.

引用本文的文献

1
Exploring Protein Misfolding in Amyotrophic Lateral Sclerosis: Structural and Functional Insights.
Biomedicines. 2025 May 9;13(5):1146. doi: 10.3390/biomedicines13051146.
2
Proteomics Analysis of the TDP-43 Interactome in Cellular Models of ALS Pathogenesis.
J Neurochem. 2025 May;169(5):e70079. doi: 10.1111/jnc.70079.
3
Spatial and Temporal Single-Cell Profiling of RNA Compartmentalization in Neurons with Nanotweezers.
ACS Nano. 2025 May 20;19(19):18522-18533. doi: 10.1021/acsnano.5c02056. Epub 2025 May 6.
6
Annexin A11 aggregation in FTLD-TDP type C and related neurodegenerative disease proteinopathies.
Acta Neuropathol. 2024 Jun 19;147(1):104. doi: 10.1007/s00401-024-02753-7.
7
Aberrant protein aggregation in amyotrophic lateral sclerosis.
J Neurol. 2024 Aug;271(8):4826-4851. doi: 10.1007/s00415-024-12485-z. Epub 2024 Jun 13.
8
Loss of TDP-43 function contributes to genomic instability in amyotrophic lateral sclerosis.
Front Neurosci. 2023 Oct 2;17:1251228. doi: 10.3389/fnins.2023.1251228. eCollection 2023.
9
Mitigating a TDP-43 proteinopathy by targeting ataxin-2 using RNA-targeting CRISPR effector proteins.
Nat Commun. 2023 Oct 14;14(1):6492. doi: 10.1038/s41467-023-42147-z.

本文引用的文献

1
Induced pluripotent stem cell modeling of multisystemic, hereditary transthyretin amyloidosis.
Stem Cell Reports. 2013 Oct 31;1(5):451-63. doi: 10.1016/j.stemcr.2013.10.003. eCollection 2013.
2
RNA toxicity from the ALS/FTD C9ORF72 expansion is mitigated by antisense intervention.
Neuron. 2013 Oct 16;80(2):415-28. doi: 10.1016/j.neuron.2013.10.015.
3
Mutations in prion-like domains in hnRNPA2B1 and hnRNPA1 cause multisystem proteinopathy and ALS.
Nature. 2013 Mar 28;495(7442):467-73. doi: 10.1038/nature11922. Epub 2013 Mar 3.
4
Accelerated disease onset with stabilized familial amyotrophic lateral sclerosis (ALS)-linked mutant TDP-43 proteins.
J Biol Chem. 2013 Feb 1;288(5):3641-54. doi: 10.1074/jbc.M112.433615. Epub 2012 Dec 12.
5
Regulated protein aggregation: stress granules and neurodegeneration.
Mol Neurodegener. 2012 Nov 20;7:56. doi: 10.1186/1750-1326-7-56.
7
Divergent roles of ALS-linked proteins FUS/TLS and TDP-43 intersect in processing long pre-mRNAs.
Nat Neurosci. 2012 Nov;15(11):1488-97. doi: 10.1038/nn.3230. Epub 2012 Sep 30.
8
NIH Image to ImageJ: 25 years of image analysis.
Nat Methods. 2012 Jul;9(7):671-5. doi: 10.1038/nmeth.2089.
9
Drug screening for ALS using patient-specific induced pluripotent stem cells.
Sci Transl Med. 2012 Aug 1;4(145):145ra104. doi: 10.1126/scitranslmed.3004052.

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